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Targeting Sulfotransferase (SULT) 2B1b as a Regulator of Cholesterol Metabolism in Prostate Cancer.

机译：靶向磺基转移酶（sULT）2B1b作为前列腺癌中胆固醇代谢的调节剂。

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作者
Ratliff, T.;
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年度 2015
页码 1-15
总页数 15
原文格式 PDF
正文语种 eng
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关键词
Cholesterol metabolism; Prostate cancer;

机译：胆固醇代谢;前列腺癌;

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专利

1. Cholesterol Sulfotransferase SULT2B1b Modulates Sensitivity to Death Receptor Ligand TNF alpha in Castration-Resistant Prostate Cancer [J] . Vickman Renee E., Yang Jiang, Lanman Nadia A., Molecular cancer research: MCR . 2019,第6期

机译：胆固醇磺基转移酶SULT2B1B在抗阉割前列腺癌中调节对死亡受体配体TNFα的敏感性
2. A key regulator of cholesterol homoeostasis, SREBP-2, can be targeted in prostate cancer cells with natural products. [J] . Krycer J. R., Phan L., Brown A. J. The Biochemical Journal . 2012,第Pta2期

机译：胆固醇稳态的关键调节剂SREBP-2可以天然产物靶向前列腺癌细胞。
3. A key regulator of cholesterol homoeostasis, SREBP-2, can be targeted in prostate cancer cells with natural products. [J] . Krycer J. R., Phan L., Brown A. J. The Biochemical Journal . 2012,第Pta2期

机译：胆固醇稳态的关键调节剂SREBP-2可以天然产物靶向前列腺癌细胞。
4. Fatty acids, cholesterol, and sphingolipids: interactive regulators and targets for SREBP processing [C] . Rebecca A. Juliano, Richard J. Deckelbaum, Tilla S. Worgall International Symposium on Atherosclerosis . 2004

机译：脂肪酸，胆固醇和鞘脂：互动调节剂和Srebp加工的目标
5. Targeting a self/tumor antigen expressed in the prostate for adoptive T cell immunotherapy of prostate cancer. [D] . Chou, Cassie K. 2012

机译：靶向前列腺中表达的自身/肿瘤抗原，用于前列腺癌的过继T细胞免疫疗法。
6. SAT-114 Loss of DHEA-Targeting SULT2b1b Sulfotransferase Exacerbates Aggressive Traits of Prostate Cancer [O] . Chung Seog Song, Sulgi Park, Shoulei Jiang, 2020

机译：SAT-114 DHEA靶向SULT2B1B磺膦酸酶的丧失加剧了前列腺癌的激进特征
7. Mutational Analysis of Human Hydroxysteroid Sulfotransferase SULT2B1 Isoforms Reveals That Exon 1B of the SULT2B1 Gene Produces Cholesterol Sulfotransferase, whereas Exon 1A Yields Pregnenolone Sulfotransferase [O] . Hirotoshi Fuda, Young C. Lee, Chikara Shimizu, 2002

机译：人羟类磺酰胺转移酶SULT2B1同种型的突变分析表明，SULT2B1基因的外显子1B产生胆固醇磺旋转转移酶，而外显子1A产生妊娠染色砜苯甲酸酯酶

Targeting Sulfotransferase (SULT) 2B1b as a Regulator of Cholesterol Metabolism in Prostate Cancer.

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