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Strategies to Counteract Resistance Mechanisms in CAR+ T-Cell-Based Immunotherapy for Triple-Negative Breast Cancer.

机译：抵抗基于CaR + T细胞的三阴性乳腺癌免疫治疗耐药机制的策略。

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Ferrone, S.;
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年度 2017
页码 1-17
总页数 17
原文格式 PDF
正文语种 eng
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1. Impact, mechanisms, and novel chemotherapy strategies for overcoming resistance to anthracyclines and taxanes in metastatic breast cancer. [J] . Perez EA Breast cancer research and treatment. . 2009,第2期

机译：在转移性乳腺癌中克服对蒽环类和紫杉烷类药物的耐药性的影响，机制和新颖的化学疗法策略。
2. Mechanisms and strategies to overcome chemotherapy resistance in metastatic breast cancer. [J] . Coley HM Cancer Treatment Reviews . 2008,第4期

机译：克服转移性乳腺癌化疗耐药的机制和策略。
3. Role of endothelin-1 in tamoxifen resistance: Mechanism for a new possible treatment strategy in breast cancer. [J] . Namazi S, Rouhani-Fard SA, Abedtash H Medical hypotheses . 2008,第1期

机译：内皮素-1在他莫昔芬耐药中的作用：一种新的乳腺癌治疗策略的机制。
4. Characterization of endogenous phosphorylation in p130Cas, an anti-estrogen resistance generating protein in human breast cancer. [C] . Giuseppe Infusini, Anthony Makkinje, David H. Perlman, ASMS Conference on Mass Spectrometry and Allied Topics . 2008

机译：P130CAs中内源性磷酸化的表征，人乳腺癌中抗雌激素产生蛋白质。
5. Regulation and action of SKP2 and RhoA in cell and tumor models: Investigation into the molecular mechanisms responsible for the aggressive phenotype of triple-negative breast cancer. [D] . Fagan-Solis, Katerina D. 2013

机译：在细胞和肿瘤模型中SKP2和RhoA的调控和作用：调查导致三阴性乳腺癌的侵袭性表型的分子机制。
6. Oxidative stress and counteracting mechanisms in hormone receptor positive triple-negative and basal-like breast carcinomas [O] . Peeter Karihtala, Saila Kauppila, Ylermi Soini, 2011

机译：激素受体阳性三阴性和基底样乳腺癌中的氧化应激及其抑制机制
7. Oxidative stress and counteracting mechanisms in hormone receptor positive, triple-negative and basal-like breast carcinomas [O] . Peeter Karihtala, Saila Kauppila, Ylermi Soini, 2011

机译：激素受体阳性，三阴性和基底样乳腺癌中的氧化应激及其抑制机制
8. Strategies to Counteract Resistance Mechanisms in CAR+ T-Cell-Based Immunotherapy for Triple-Negative Breast Cancer. [R] . Dotti , G. 2017

机译：抵抗基于CaR + T细胞的三阴性乳腺癌免疫治疗耐药机制的策略。

Strategies to Counteract Resistance Mechanisms in CAR+ T-Cell-Based Immunotherapy for Triple-Negative Breast Cancer.

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