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Does the Loss of Stromal Caveolin-1 Remodel the Tumor Microenvironment by Activating Src-Mediated PEAK1 and PI3K Pathways.

机译：基质Caveolin-1的缺失是否通过激活src介导的pEaK1和pI3K途径重塑肿瘤微环境。

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作者
Sobreiro, M. R.; Yang, W.; Di Vizio, D.; Freeman, M. R.;
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年度 2017
页码 1-55
总页数 55
原文格式 PDF
正文语种 eng
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关键词
Prostate cancer; Kinases; Metastasis; Fibroblasts; Cell membrane; Ev(extracellular vesicles); Pi3k;

机译：前列腺癌;激酶;转移;成纤维细胞;细胞膜; Ev（细胞外囊泡）; pi3k;
入库时间 2022-08-29 10:46:08

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专利

1. Biomechanical remodeling of the microenvironment by stromal caveolin-1 favors tumor invasion and metastasis [J] . Goetz J.G., Minguet S., Navarro-Lérida I., Cell . 2011,第1期

机译：间质caveolin-1对微环境的生物力学重塑有利于肿瘤的侵袭和转移
2. Loss of stromal caveolin-1 leads to oxidative stress, mimics hypoxia and drives inflammation in the tumor microenvironment, conferring the "reverse Warburg effect": A transcriptional informatics analysis with validation. [J] . Pavlides S, Vera I, Frank PG, Cell cycle . 2010,第11期

机译：基质小窝蛋白-1的缺失会导致氧化应激，模拟缺氧并在肿瘤微环境中引发炎症，从而赋予“逆向Warburg效应”：经过验证的转录信息学分析。
3. Loss of stromal caveolin-1 leads to oxidative stress, mimics hypoxia and drives inflammation in the tumor microenvironment, conferring the "reverse Warburg effect [J] . Pavlides S, Vera I, Frank PG, Cell cycle . 2010,第11期

机译：丧失基质Caveolin-1导致氧化应激，模仿缺氧和驱动肿瘤微环境中的炎症，赋予“逆转Warburg效应
4. Label-free Quantitative Proteomics of Biopsy Tissue from Breast Cancer Patients Reveals Inflammatory Activation of the Tumor Microenvironment [C] . Groessl M., Gloessmann K., Pfeiler G., American Society for Mass Spectrometry Conference on Mass Spectrometry and Allied Topics . 2013

机译：免标记的乳腺癌患者活组织检查组织的定量蛋白质组学表现出肿瘤微环境的炎症激活
5. The effect of cellular and acellular components of the tumor microenvironment on both tumor and stromal cell phenotypes in a breast cancer model system [D] . McLane, Joshua 2015

机译：乳腺癌模型系统中肿瘤微环境的细胞和非细胞成分对肿瘤和基质细胞表型的影响
6. Replication Study: Biomechanical remodeling of the microenvironment by stromal caveolin-1 favors tumor invasion and metastasis [O] . Mee Rie Sheen, Jennifer L Fields, Brian Northan, 2019

机译：复制研究：通过基质Caveolin-1的微环境进行生物力学重塑 - 1抗肿瘤侵袭和转移
7. Replication Study: Biomechanical remodeling of the microenvironment by stromal caveolin-1 favors tumor invasion and metastasis [O] . Mee Rie Sheen, Jennifer L Fields, Brian Northan, 2019

机译：复制研究：通过基质Caveolin-1的微环境进行生物力学重塑 - 1抗肿瘤侵袭和转移
8. Does the Loss of Stromal Caveolin-1 Remodel the Tumor Microenvironment by Activating Src-Mediated PEAK1 and PI3K Pathways. [R] . Sobreiro, M. R., Yang, W., DiVizio, D., 2016

机译：基质Caveolin-1的缺失是否通过激活src介导的pEaK1和pI3K途径重塑肿瘤微环境。

Does the Loss of Stromal Caveolin-1 Remodel the Tumor Microenvironment by Activating Src-Mediated PEAK1 and PI3K Pathways.

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