Potassium Deficiency in Rats: Effects on Acute Thermal Tolerance. (Reannouncementwith New Availability Information)

摘要

In recent years we have quantitated the effects of various factors which had beenreported to compromise or augment (after 'compromise') physical or thermoregulatory performance during work in the heat in the rat model of human heat injury. In many of these studies the mechanisms responsible for either the reduced or increased performance appeared self-evident, e.g. increased heat storage or heat production, decreased peripheral heat dissipation, or elevated cardiovascular stability due to expanded plasma volume. In other cases, however, the mechanisms were not so apparent and were characterized by subtle differences in the characteristic responses to heat of such circulating indices of heat injury as lactic acid, glucose, enzyme levels, and potassium (K+).