A CD1d-dependent lipid antagonist to NKT cells ameliorates atherosclerosis in ApoE(-/-) mice by reducing lesion necrosis and inflammation

Li Yi; Kanellakis Peter; Hosseini Hamid; Cao Anh; Deswaerte Virginie; Tipping Peter; Toh Ban-Hock; Bobik Alex; Kyaw Tin

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A CD1d-dependent lipid antagonist to NKT cells ameliorates atherosclerosis in ApoE(-/-) mice by reducing lesion necrosis and inflammation

机译：依赖CD1d的NKT细胞脂质拮抗剂可通过减少病变坏死和炎症改善ApoE（-/-）小鼠的动脉粥样硬化

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Aims Atherosclerosis-related deaths from heart attacks and strokes remain leading causes of global mortality, despite the use of lipid-lowering statins. Thus, there is an urgent need to develop additional therapies.

机译：目的尽管使用降脂他汀类药物，心脏病发作和中风引起的与动脉粥样硬化相关的死亡仍然是全球死亡率的主要原因。因此，迫切需要开发其他疗法。

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《Cardiovascular Research》 |2016年第2期|共13页
作者
Li Yi; Kanellakis Peter; Hosseini Hamid; Cao Anh; Deswaerte Virginie; Tipping Peter; Toh Ban-Hock; Bobik Alex; Kyaw Tin;
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正文语种 eng
中图分类心脏、血管（循环系）疾病;
关键词
NKT cells; Atherosclerosis; ApoE(-/-) mice; DPPE-PEG; NKT antagonist;

机译：NKT细胞;动脉粥样硬化;ApoE（-/-）小鼠;DPPE-PEG;NKT拮抗剂;

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专利

1. A CD1d-dependent lipid antagonist to NKT cells ameliorates atherosclerosis in ApoE(-/-) mice by reducing lesion necrosis and inflammation [J] . Li Yi, Kanellakis Peter, Hosseini Hamid, Cardiovascular Research . 2016,第2期

机译：依赖CD1d的NKT细胞脂质拮抗剂可通过减少病变坏死和炎症改善ApoE（-/-）小鼠的动脉粥样硬化
2. Medium-, long- and medium-chain-type structured lipids ameliorate high-fat diet-induced atherosclerosis by regulating inflammation, adipogenesis, and gut microbiota in ApoE(-/-)mice [J] . Food & Function . 2020,第6期

机译：中，长链和中链型结构化脂质通过调节炎症，脂肪发生和肠道微生物在ApoE（/ - ）小鼠中来改善高脂肪饮食诱发的动脉粥样硬化
3. 5-lipoxygenase deficiency reduces hepatic inflammation and tumor necrosis factor alpha-induced hepatocyte damage in hyperlipidemia-prone ApoE-null mice. [J] . Martinez Clemente M, Ferre N, Gonzalez Periz Hepatology: Official Journal of the American Association for the Study of Liver Diseases . 2010,第3期

机译：5-脂氧合酶缺乏症可降低易发高脂血症的ApoE无效小鼠的肝脏炎症和肿瘤坏死因子α诱导的肝细胞损伤。
4. The role of experimental model of atherosclerosis: apoE-knockout mice in developing new drugs against atherosclerosis [C] . Jawien J. International Congress on Coronary Artery Disease. . 2013

机译：动脉粥样硬化的实验模型的作用：Apoe-Nockout小鼠在制育新药物对动脉粥样硬化的作用
5. Arsenic in drinking water causes gene expression changes in the liver related to inflammation and metabolic dysfunction and accelerates atherosclerosis in APOE-/- mice. [D] . Zajack, Matthew R. 2010

机译：饮用水中的砷会导致肝脏中与炎症和代谢功能异常有关的基因表达变化，并加速APOE-/-小鼠的动脉粥样硬化。
6. Astragalus Flavone Ameliorates Atherosclerosis and Hepatic Steatosis Via Inhibiting Lipid-Disorder and Inflammation in apoE−/− Mice [O] . Chuanrui Ma, Jing Zhang, Shu Yang, 2020

机译：黄芪黄酮通过抑制脂质障碍和肝脏炎症和肝脏粥样硬化和肝脏脂肪变化而改善了Apoe - / - 小鼠的炎症
7. A CD1d-dependent lipid antagonist to NKT cells ameliorates atherosclerosis in ApoE−/−mice by reducing lesion necrosis and inflammation [O] . Yi Li, Peter Kanellakis, Hamid Hosseini, 2015

机译：通过减少病变坏死和炎症，将CD1D依赖性脂质拮抗剂改善了apoe - / - 小鼠的动脉粥样硬化

A CD1d-dependent lipid antagonist to NKT cells ameliorates atherosclerosis in ApoE(-/-) mice by reducing lesion necrosis and inflammation

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