首页> 外文期刊>Bulletin of experimental biology and medicine >Ca(2+) Transport into Sarcoplasmic Reticulum and Immediate-Early Response Proteins in the Myocardium of Rats Resuscitated after Systemic Circulatory Arrest.
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Ca(2+) Transport into Sarcoplasmic Reticulum and Immediate-Early Response Proteins in the Myocardium of Rats Resuscitated after Systemic Circulatory Arrest.

机译:Ca(2+)运输到肌浆网和系统循环骤停复苏的大鼠心肌中的立即反应蛋白。

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摘要

Activity of antioxidant defense enzymes and content of stress protein HSP70 in the heart increased in passive and, to a lesser extent, in active rats on day 7 of the postresuscitation period after systemic circulatory arrest. The resistance of membrane structures in the heart to endogenous damaging factors in passive rats was lower than in active animals. The degree of compensation in active rats was much higher than in passive animals at these terms of the postresuscitation period.
机译:在系统循环停止后第7天,被动活动的心脏中抗氧化剂防御酶的活性和应激蛋白HSP70的含量在被动状态下有所增加,而活动大鼠的程度较小。被动大鼠心脏中的膜结构对内源性破坏因子的抵抗力低于主动动物。在复苏后的这段时间内,主动大鼠的补偿程度远高于被动动物。

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