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A case report of acute myocardial infarction induced by coronary spasm.

机译:冠状动脉痉挛致急性心肌梗死一例报告。

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摘要

A 53-year-old male complaining of chest pain was admitted to our hospital with suspected acute myocardial infarction (AMI). Emergent coronary angiography (CAG) determined a totally occluded middle right coronary artery (RCA). Thrombus aspiration was conducted, followed by intravascular ultrasound (IVUS) imaging. Diffuse intima plus media thickness was identified at the obstruction site and a thrombus was observed proximally to the occlusion site on IVUS. Following isosorbide dinitrate (ISDN) administration, dilatation of the RCA was confirmed. IVUS study indicated the luminal dilatation was achieved by the release of the diffuse intima plus media thickening. Of note, plaque volume showed no significant difference after administration of ISDN at any vessel site. These results clearly show that luminal dilatation and vessel dilatation were achieved from the redistribution of plaque volume (intima plus media). A follow-up CAG showed no significant stenosis in the RCA. After a provocation test using methylergometrine maleate, the RCA was totally occluded at the very site of the initial event. The involvement of vasospasm as a cause of AMI in the present case was doubly confirmed with characteristic IVUS images of vasospasm in the acute phase and with a provocation test at follow-up.
机译:一名53岁的主诉胸痛的男性因怀疑患有急性心肌梗塞(AMI)而入院。紧急冠状动脉造影(CAG)确定了完全闭塞的右中冠状动脉(RCA)。进行血栓抽吸,然后进行血管内超声(IVUS)成像。在梗阻部位发现弥漫性内膜和介质厚度,在IVUS闭塞部位近端观察到血栓。硝酸异山梨酯(ISDN)给药后,证实RCA扩张。 IVUS研究表明,腔内扩张是通过弥漫性内膜的释放和介质的增厚来实现的。值得注意的是,在任何血管部位使用ISDN后,斑块体积均无显着差异。这些结果清楚地表明,通过斑块体积(内膜加培养基)的重新分布实现了管腔扩张和血管扩张。随访的CAG显示RCA无明显狭窄。在使用马来酸麦角麦角新碱进行激发试验后,RCA在最初事件的现场完全被阻塞。在本例中,血管痉挛是导致AMI的原因,在急性期通过血管痉挛的特征性IVUS图像和后续的激发试验双重证实。

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