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首页> 外文期刊>Nature Metabolism >Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (vol 1, pg 958, 2019)
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Defects in mtDNA replication challenge nuclear genome stability through nucleotide depletion and provide a unifying mechanism for mouse progerias (vol 1, pg 958, 2019)

机译:核mtDNA复制缺陷挑战通过核苷酸枯竭和基因组的稳定性提供了一种统一的机制鼠标早衰症(1卷,958 pg, 2019)

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