机译:雌激素促进LNCRNA H19表达,以调节BMSCs的骨质发生分化,通过MIR-532-3P / SIRT1轴减少骨质疏松症
Huazhong Univ Sci &
Technol Union Hosp Tongji Med Coll Dept Obstet &
Gynecol 1277 JieFang Ave;
Zhengzhou Univ Dept Urol Surg Henan Prov Peoples Hosp Zhengzhou 450003 Henan Peoples R China;
Huazhong Univ Sci &
Technol Union Hosp Tongji Med Coll Dept Pancreat Surg Wuhan 430022 Hubei;
Huazhong Univ Sci &
Technol Union Hosp Tongji Med Coll Dept Obstet &
Gynecol 1277 JieFang Ave;
Huazhong Univ Sci &
Technol Union Hosp Tongji Med Coll Dept Obstet &
Gynecol 1277 JieFang Ave;
Estrogen; lncRNA H19; miR-532-3p; Osteogenic differentiation; Osteoporosis;
机译:肥胖症调节MiR-467 / Hoxa10轴上的骨质发生分化和BMSC衍生外来辐射术的骨折愈合
机译:MicroRNA-19B-3P通过与LNCRNA H19相互作用来促进BMSCs的细胞增殖和骨质发生分化
机译:重组腺病毒(Adeasy System)介导的长非编码RNA H19(LNCRNA H19)的外源表达双相调节间充质干细胞(MSC)的骨质发生
机译:Prox1和成纤维细胞生长因子受体形成新的调节环,促进晶状体纤维分化并调节全局基因表达
机译:肥胖症调节miR-467 / hoxa10轴上的骨质化分化和bmsc衍生外来辐射术的骨折愈合
机译:肥胖症调节miR-467 / hoxa10轴上的骨质化分化和bmsc衍生外来辐射术的骨折愈合