机译:Beta-Arectrin 2淬火TLR信令以促进EPEC的免疫逃避
Fudan Univ Dept Immunol Sch Basic Med Sci Shanghai 200032 Peoples R China;
Fudan Univ Dept Immunol Sch Basic Med Sci Shanghai 200032 Peoples R China;
Fudan Univ Dept Immunol Sch Basic Med Sci Shanghai 200032 Peoples R China;
Fudan Univ Dept Immunol Sch Basic Med Sci Shanghai 200032 Peoples R China;
Third Mil Med Univ Coll Pharm Natl Engn Res Ctr Immunol Prod Dept Microbiol &
Biochem Pharm;
Tongji Univ Shanghai Key Lab Signaling &
Dis Res Lab Receptor Based Biomed Collaborat Innovat;
Shanghai Jiao Tong Univ Sch Publ Hlth Sch Med Shanghai Peoples R China;
Fudan Univ Sch Basic Med Sci Dept Physiol &
Pathophysiol Shanghai Peoples R China;
Bar Ilan Univ Mina &
Everard Goodman Fac Life Sci Bar Ilan Inst Nanotechnol &
Adv Mat Ramat Gan;
Capital Med Univ Beijing Hosp Tradit Chinese Med Beijing Peoples R China;
Fudan Univ Dept Immunol Sch Basic Med Sci Shanghai 200032 Peoples R China;
Fudan Univ Dept Immunol Sch Basic Med Sci Shanghai 200032 Peoples R China;
beta-arrestin 2; TRAF6; TAK1; TLR signaling; immune evasion; EPEC;
机译:单一细菌免疫规避策略可消除TLR4下游的MyD88和TRIF信号通路
机译:疟疾寄生虫需要TLR9信号通过激活调节性T细胞逃逸免疫。
机译:TGF beta信号在胰腺肿瘤微环境中促进纤维化和免疫逃逸,以促进肿瘤发生。
机译:VIP对TNBS诱导性结肠炎淋巴结免疫细胞TLR2和TLR4表达的影响
机译:SARS冠状病毒拮抗RIG-1启动的先天免疫信号传导,但通过衔接子分子TRIF被TLR信号传导识别
机译:单一细菌免疫逃避策略可消除TLR4下游的MyD88和TRIF信号通路
机译:β-arrierin 2淬火TLR信令以促进EPEC的免疫逃避
机译:先天免疫系统TLR受体识别病原体的结构基础