机译:通过PI3K / AKT / MTOR途径在Rotenone诱导的帕金森病大鼠的早期和中等阶段通过自噬介导的氢饱和盐水通过自噬介导
Shandong First Med Univ &
Shandong Acad Med Sci Dept Physiol Basic Med Coll Tai An 271000;
Taian City Cent Hosp Dept Nephrol Tai An 271000 Shandong Peoples R China;
Taian City Cent Hosp Postdoctoral Workstn Tai An 271000 Shandong Peoples R China;
Shandong First Med Univ &
Shandong Acad Med Sci Life Sci Res Ctr Tai An 271000 Shandong Peoples;
Taian Tradit Chinese Med Hosp Dept Electrocardiogram Tai An 271000 Shandong Peoples R China;
Shandong First Med Univ &
Shandong Acad Med Sci Dept Physiol Basic Med Coll Tai An 271000;
Shandong First Med Univ &
Shandong Acad Med Sci Dept Physiol Basic Med Coll Tai An 271000;
Shandong First Med Univ &
Shandong Acad Med Sci Dept Physiol Basic Med Coll Tai An 271000;
Shandong First Med Univ &
Shandong Acad Med Sci Life Sci Res Ctr Tai An 271000 Shandong Peoples;
Autophagy; Catecholaminergic neurons; Early and medium stages; Hydrogen-rich saline; Parkinson's disease; Rostral ventrolateral medulla;
机译:羊角酮诱导帕金森病的神经保护作用在大鼠中:PI3K / AKT / MTOR信号通路与MiRNA-7和MiRNA-221的增强表达之间的相互作用
机译:PI3K / AKT / MTOR途径通过Dexmedetomidine参与神经保护术抑制大鼠创伤性脑损伤后神经源性自噬
机译:艾灸对马松酮诱导帕金森病模型大鼠MTOR介导的自噬的影响
机译:阳离子聚苯乙烯纳米球通过抑制Akt / mTOR和激活巨噬细胞和上皮细胞中的AMPK信号通路来诱导自噬
机译:PI3k / AKT / mTOR信号通路的遗传分析。
机译:自噬受损对急性实验性帕金森病的反应触发炎症小体:PI3K / Akt / mTOR途径的参与
机译:PI3K / AKT / MTOR途径通过Dexmedetomidine参与神经保护术抑制大鼠创伤性脑损伤后神经源性自噬