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机译:胜迈注射通过调制AMPK,MTOR和JNK途径衰减脑缺血/再灌注诱导的自噬
China Pharmaceut Univ Dept Complex Prescript TCM Jiangsu Key Lab TCM Evaluat &
Translat Res 639;
China Pharmaceut Univ Dept Complex Prescript TCM Jiangsu Key Lab TCM Evaluat &
Translat Res 639;
China Pharmaceut Univ Dept Complex Prescript TCM Jiangsu Key Lab TCM Evaluat &
Translat Res 639;
China Pharmaceut Univ Dept Complex Prescript TCM Jiangsu Key Lab TCM Evaluat &
Translat Res 639;
China Pharmaceut Univ Dept Complex Prescript TCM Jiangsu Key Lab TCM Evaluat &
Translat Res 639;
China Pharmaceut Univ Dept Complex Prescript TCM Jiangsu Key Lab TCM Evaluat &
Translat Res 639;
China Pharmaceut Univ Dept Complex Prescript TCM Jiangsu Key Lab TCM Evaluat &
Translat Res 639;
China Pharmaceut Univ Dept Complex Prescript TCM Jiangsu Key Lab TCM Evaluat &
Translat Res 639;
China Pharmaceut Univ Dept Complex Prescript TCM Jiangsu Key Lab TCM Evaluat &
Translat Res 639;
Cerebral ischemia/reperfusion; neuroprotection; signalling pathways;
机译:生脉注射液通过调节AMPK,mTOR和JNK途径减轻脑缺血/再灌注诱导的自噬
机译:四种活性化合物的组合可减轻脑缺血再灌注损伤,并抑制自噬并调节AMPK / mTOR和JNK途径
机译:通过增强自噬>通过斜体> AMPK-MTOR-P70S6K途径来抑制脑缺血再灌注损伤脑缺血再灌注损伤
机译:阳离子聚苯乙烯纳米球通过抑制Akt / mTOR和激活巨噬细胞和上皮细胞中的AMPK信号通路来诱导自噬
机译:脑缺血和再灌注损伤:己酮可可碱对白细胞和白细胞的调节作用。
机译:丁香酚通过增强自噬通过AMPK-mTOR-P70S6K途径减轻脑缺血-再灌注损伤
机译:胜迈注射通过调制AMPK,MTOR和JNK途径衰减脑缺血/再灌注诱导的自噬