CFTR ADENYLATE KINASE ACTIVITY IS REQUIRED FOR NORMAL CFTR CHANNEL FUNCTION IN HUMAN AIRWAY EPITHELIA

Dong Q.; Shah V. S.; Tang X.; Welsh M.; Randak C. O.

首页> 外文期刊>Pediatric Pulmonology >CFTR ADENYLATE KINASE ACTIVITY IS REQUIRED FOR NORMAL CFTR CHANNEL FUNCTION IN HUMAN AIRWAY EPITHELIA

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CFTR ADENYLATE KINASE ACTIVITY IS REQUIRED FOR NORMAL CFTR CHANNEL FUNCTION IN HUMAN AIRWAY EPITHELIA

机译：CFTR腺苷酸激酶活性是人类气道上皮内的正常CFTR通道功能所必需的

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来源
《Pediatric Pulmonology》 |2016年第45期|共1页
作者
Dong Q.; Shah V. S.; Tang X.; Welsh M.; Randak C. O.;
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作者单位

Univ Iowa Stead Family Dept Pediat Iowa City IA USA;

Univ Iowa Dept Mol Physiol &

Biophys Iowa City IA USA;

Howard Hughes Med Inst Iowa City IA USA;

Univ Iowa Dept Mol Physiol &

Biophys Iowa City IA USA;

Univ Iowa Stead Family Dept Pediat Iowa City IA USA;

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正文语种 eng
中图分类儿科学;
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入库时间 2022-08-20 06:04:28

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专利

1. CFTR ADENYLATE KINASE ACTIVITY IS REQUIRED FOR NORMAL CFTR CHANNEL FUNCTION IN HUMAN AIRWAY EPITHELIA [J] . Dong Q., Shah V. S., Tang X., Pediatric Pulmonology . 2016,第Suppla51期

机译：人体气道上皮细胞的正常CFTR通道功能需要CFTR腺苷酸激酶活性
2. CFTR ADENYLATE KINASE ACTIVITY IS REQUIRED FOR NORMAL CFTR CHANNEL FUNCTION IN AIRWAY AND INTESTINAL EPITHELIA [J] . Dong Q., Shah V. S., Tang X., Pediatric Pulmonology . 2018,第Suppla2期

机译：CFTR腺苷酸激酶活性是气道和肠上皮内的正常CFTR通道功能
3. CFTR ADENYLATE KINASE ACTIVITY IS REQUIRED FOR NORMAL CFTR CHANNEL FUNCTION IN AIRWAY AND INTESTINAL EPITHELIA [J] . Dong Q., Shah V. S., Tang X., Pediatric Pulmonology . 2018,第Suppla2期

机译：CFTR腺苷酸激酶活性是气道和肠上皮内的正常CFTR通道功能
4. Multifaceted activities of class Ⅰ phosphatidylinositol 3-kinase inhibitor ZSTK474 on human breast cancer cells [C] . Xin Peng, Zhengming Wang, Chang Zhou, International Forum on Leading Edge Technologies on Crystallization Engineering and Pharmaceutics Development;Tianjin University;Tianjin Medical University . -1

机译：Ⅰ类磷脂酰肌醇3-激酶抑制剂ZSTK474对人乳腺癌细胞的多丝活动
5. Coexpression of mutant forms of CFTR inhibit wild-type CFTR expression and function in airway epithelium. [D] . Tucker, Torry Alle. 2004

机译：CFTR突变形式的共表达抑制气道上皮中的野生型CFTR表达和功能。
6. Mutating the Conserved Q-loop Glutamine 1291 Selectively Disrupts Adenylate Kinase-dependent Channel Gating of the ATP-binding Cassette (ABC) Adenylate Kinase Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) and Reduces Channel Function in Primary Human Airway Epithelia [O] . Qian Dong, Sarah E. Ernst, Lynda S. Ostedgaard, 2015

机译：突变保守的Q环谷氨酰胺1291选择性破坏ATP结合盒（ABC）的腺苷酸激酶依赖性通道门控腺苷酸激酶囊性纤维化跨膜电导调节剂（CFTR）并减少主要人气道上皮细胞的通道功能
7. Mutating the Conserved Q-loop Glutamine 1291 Selectively Disrupts Adenylate Kinase-dependent Channel Gating of the ATP-binding Cassette (ABC) Adenylate Kinase Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) and Reduces Channel Function in Primary Human Airway Epithelia [O] . Qian Dong, Sarah E. Ernst, Lynda S. Ostedgaard, 2015

机译：突变保守的Q环谷氨酰胺1291选择性地破坏ATP结合盒（ABC）腺苷酸激酶囊性纤维化纤维化纤维化纤维化型纤维化型纤维化常规调节器（CFTR）的腺苷酸激酶依赖性通道门，并减少了原发性人体气道上皮内的信道功能
8. Methods for Modulating PPAR Biological Activity for the Treatment of Diseases Caused By Mutations in the CFTR Gene. [R] . Freedman, S. D. 2005

机译：调节ppaR生物活性的方法用于治疗由CFTR基因突变引起的疾病。

CFTR ADENYLATE KINASE ACTIVITY IS REQUIRED FOR NORMAL CFTR CHANNEL FUNCTION IN HUMAN AIRWAY EPITHELIA

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