机译:miR-200b通过靶向Akt途径来抑制CD133(+)胶质瘤细胞
Jinan Univ Dept Neurol Guangzhou 510632 Guangdong Peoples R China;
Guangdong Pharmaceut Univ Affiliated Hosp 1 Sch Clin Med Dept Neurol 16 Jichang Load Guangzhou;
Guangdong Pharmaceut Univ Affiliated Hosp 1 Sch Clin Med Dept Neurol 16 Jichang Load Guangzhou;
Guangdong Pharmaceut Univ Affiliated Hosp 1 Sch Clin Med Dept Neurol 16 Jichang Load Guangzhou;
Guangdong Pharmaceut Univ Affiliated Hosp 1 Sch Clin Med Dept Neurol 16 Jichang Load Guangzhou;
Guangdong Pharmaceut Univ Affiliated Hosp 1 Sch Clin Med Dept Neurol 16 Jichang Load Guangzhou;
Guangdong Pharmaceut Univ Affiliated Hosp 1 Sch Clin Med Dept Neurosurg Guangzhou 510080;
Guangdong Pharmaceut Univ Affiliated Hosp 1 Sch Clin Med Dept Neurol 16 Jichang Load Guangzhou;
机译:miR-200b通过靶向Akt途径来抑制CD133(+)胶质瘤细胞
机译:Micro RNA RNA -21促进胶质瘤细胞增殖并通过PTEN PTEN / PI PI PI 3K / AKT AKT信号通路靶向Spry Spry 1来抑制衰老和凋亡
机译:MiR-489抑制增殖,细胞周期进展,并通过靶向Spin1介导的PI3K / AKT途径诱导胶质瘤细胞的凋亡
机译:作为新的支架涂层材料,吉非替尼特异性地通过抑制EGFR / AKT途径磷酸化来抑制平滑的肌细胞增殖
机译:多靶点受体酪氨酸激酶抑制剂利尼法尼(ABT-869)通过磷酸肌醇3激酶(PI3K)/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
机译:miR-200b通过靶向AKT途径抑制CD133 +胶质瘤细胞
机译:收缩陈述:MicroRNA-21通过PTEN / PI3K / AKT信号通路靶向SPRY1来促进胶质瘤细胞增殖并抑制衰老和细胞凋亡