...
机译:E3泛素连接酶瘙痒和YAP1具有拮抗作用在ASPP2蛋白稳定性的调节中
Fudan Univ Sch Life Sci State Key Lab Genet Engn Shanghai 200433 Peoples R China;
Fudan Univ Sch Life Sci State Key Lab Genet Engn Shanghai 200433 Peoples R China;
Fudan Univ Sch Life Sci State Key Lab Genet Engn Shanghai 200433 Peoples R China;
Fudan Univ Sch Life Sci State Key Lab Genet Engn Shanghai 200433 Peoples R China;
Shanghai Jiao Tong Univ Sch Med Shanghai Peoples Hosp 1 Dept Urol Shanghai 200080 Peoples R;
Shanghai Jiao Tong Univ Sch Med Shanghai Peoples Hosp 1 Dept Urol Shanghai 200080 Peoples R;
Fudan Univ Sch Life Sci State Key Lab Genet Engn Shanghai 200433 Peoples R China;
Fudan Univ Sch Life Sci State Key Lab Genet Engn Shanghai 200433 Peoples R China;
Fudan Univ Sch Life Sci State Key Lab Genet Engn Shanghai 200433 Peoples R China;
Fudan Univ Sch Life Sci State Key Lab Genet Engn Shanghai 200433 Peoples R China;
Ubiquitination; Proteasomal degradation; WW domain; PPXY motif; Apoptosis;
机译:E3泛素连接酶Itch和Yap1在调节ASPP2蛋白稳定性中具有拮抗作用
机译:E3泛素连接酶瘙痒和YAP1具有拮抗作用在ASPP2蛋白稳定性的调节中
机译:E3泛素连接酶Itch和Yap1在调节ASPP2蛋白稳定性中具有拮抗作用
机译:E3泛素连接酶CBL-B通过抑制PI3K / AKT信号通路和P-糖蛋白下调逆转MCF-7 / ADR细胞的多药耐药性
机译:痒E3泛素连接酶调节LATS1肿瘤抑制因子的稳定性
机译:E3泛素连接酶Itch以乙酰化依赖性方式调节肿瘤抑制蛋白RASSF5 / NORE1的稳定性
机译:E3泛素连接酶Itch和Yap1在调节ASPP2蛋白稳定性中具有拮抗作用