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机译:LNCAPC LNCAPC通过EzH2介导的APC转录抑制驱动WNT /β-Catenin活化和肝脏TIC自我更新
Department of Histology and EmbryologyCollege of Basic Medicine Zhengzhou UniversityHenan China;
Department of Otolaryngology Medical SchoolUniversity of Minnesota Twin Cities CampusMinneapolis;
Department of PathologySchool of Medicine University of VirginiaVirginia;
Department of Nuclear Medicine The Affiliated Beijing Anzhen Hospital of Capital Medical;
Department of Hepatopancreatobiliary Surgery The Affiliated Cancer Hospital of Zhengzhou;
Department of Cancer Biology Immunotherapy The Affiliated Cancer Hospital of Zhengzhou;
Department of Cancer Biology Immunotherapy The Affiliated Cancer Hospital of Zhengzhou;
Department of Histology and EmbryologyCollege of Basic Medicine Zhengzhou UniversityHenan China;
Department of Histology and EmbryologyCollege of Basic Medicine Zhengzhou UniversityHenan China;
liver cancer; long noncoding RNA; remodeling complex; tumor initiating cells; Wnt/β‐catenin;
机译:LNCAPC LNCAPC通过EzH2介导的APC转录抑制驱动WNT /β-Catenin活化和肝脏TIC自我更新
机译:S.6.1β-catenin是SScS的中央介体.6.2 JAK2是TGF-β对成纤维细胞活化和组织纤维化的刺激作用的新型介体.6.3TGF-β激活经典WNT信号驱动SScS.6.4抑制纤维化。的
机译:LNCTIC1与β-catenin相互作用以驱使肝脏自我更新和肝脏肿瘤瘤
机译:转录共激活因子CoCoA在Wnt /β-catenin信号通路中的作用:CoCoA N末端激活域的分子机制
机译:LncFZD6通过BRG1介导的FZD6转录激活启动Wnt /β-catenin和肝脏TIC自我更新
机译:LNCFZD6通过BRG1介导的FZD6转录激活启动WNT /β-Catenin和肝脏TIC自我更新