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机译:暴露于氯菊酯促进雄性C57BL / 6J小鼠中的高脂肪饮食诱导的体重增加和胰岛素抗性
Department of Food Science University of Massachusetts;
Department of Food Science University of Massachusetts;
Department of Food Science University of Massachusetts;
Department of Food Science University of Massachusetts;
Department of Food Science University of Massachusetts;
Department of Mathematics and Statistics University of Massachusetts;
Department of Biological Sciences and Environmental Sciences Program Southern Illinois University;
Department of Veterinary and Animal Sciences University of Massachusetts;
Department of Food Science University of Massachusetts;
Permethrin; Insecticide; Obesity; Glucose metabolism;
机译:暴露于氯菊酯促进雄性C57BL / 6J小鼠中的高脂肪饮食诱导的体重增加和胰岛素抗性
机译:高脂饮食诱导的C57BL / 6J小鼠胰岛素抵抗后,胰岛淀粉样多肽(淀粉样蛋白)和胰岛素mRNA表达的差异性变化。
机译:通过增强胰岛素抵抗力并降低雌性C57BL / 6J小鼠的胰岛素抗性,改变葡萄糖代谢和高脂饮食。
机译:苯并噻唑衍生物在长期高脂肪喂养C57BL / 6J小鼠中加速了总甾醇的排泄
机译:长期饮食锌处理对饮食性肥胖C57BL / 6J小鼠糖尿病指数和脂肪组织磷脂和甘油三酸酯脂肪酸组成的影响
机译:氯菊酯的暴露促进高脂饮食诱导的雄性C57BL / 6J小鼠体重增加和胰岛素抵抗
机译:暴露于氯菊酯促进雄性C57BL / 6J小鼠中的高脂肪饮食诱导的体重增加和胰岛素抗性