机译:Oroxindin通过抑制TXNIP依赖性NF-Kappa B途径抑制DSS诱导的DSS诱导的溃疡性结肠炎中的巨噬细胞NLRP3炎症活化
Guangzhou Univ Chinese Med Sch Pharmaceut Sci Guangzhou 510006 Peoples R China;
Guangzhou Univ Chinese Med Sch Pharmaceut Sci Guangzhou 510006 Peoples R China;
Guangzhou Univ Chinese Med Affiliated Hosp 1 Guangzhou 510405 Peoples R China;
Guangzhou Univ Chinese Med Sch Pharmaceut Sci Guangzhou 510006 Peoples R China;
Guangzhou Univ Chinese Med Sch Pharmaceut Sci Guangzhou 510006 Peoples R China;
Guangzhou Univ Chinese Med Sch Pharmaceut Sci Guangzhou 510006 Peoples R China;
Guangzhou Univ Chinese Med Sch Pharmaceut Sci Guangzhou 510006 Peoples R China;
Guangzhou Univ Chinese Med Sch Pharmaceut Sci Guangzhou 510006 Peoples R China;
Guangzhou Univ Chinese Med Sch Pharmaceut Sci Guangzhou 510006 Peoples R China;
Guangdong Pharmaceut Univ Sch Chinese Med Guangzhou 510006 Peoples R China;
Huazhong Univ Sci &
Technol Tongji Med Coll Affiliated Puai Hosp Dept Lab Med Wuhan 430030;
Guangzhou Univ Chinese Med Sch Pharmaceut Sci Guangzhou 510006 Peoples R China;
Guangzhou Univ Chinese Med Sch Pharmaceut Sci Guangzhou 510006 Peoples R China;
ulcerative colitis; oroxindin; NLRP3 inflammasome; TXNIP; NF-kappa B signaling; anti-inflammatory;
机译:Oroxindin通过抑制TXNIP依赖性NF-Kappa B途径抑制DSS诱导的DSS诱导的溃疡性结肠炎中的巨噬细胞NLRP3炎症活化
机译:SINIGROM通过抑制NF-Kappa B / MAPK途径或NLRP3炎症在巨噬细胞中的NLRP3发炎物激活来抑制炎症介质的产生
机译:libertellenone m,源自内生真菌Phomopsis sp的二萜。 S12,通过抑制NF-Kappa B和NLRP3炎性组活化的核转移来保护DSS诱导的结肠炎
机译:补体蛋白C1q调节巨噬细胞NLRP3炎性体激活。
机译:Oroxindin通过抑制TXNip依赖性NF-κB途径抑制DSS诱导的DSS诱导的溃疡性结肠炎中的巨噬细胞NLRP3炎性激活
机译:通过降低氧化应激,通过抑制巨噬细胞的Txnip依赖性NLRP3炎性激活来保护FlaVOnoid VI-16免受DSS诱导的结肠炎。