机译:CDCA7的过度表达预测预后差和诱导ezH2介导的三阴性乳腺癌进展
State Key Laboratory of Oncology in South China Collaborative Innovation Center for Cancer;
State Key Laboratory of Oncology in South China Collaborative Innovation Center for Cancer;
Department of RadiotherapyThe Affiliated Yantai Yuhuangding Hospital of Qingdao UniversityYantai;
Binzhou Medical UniversityYantai China;
State Key Laboratory of Oncology in South China Collaborative Innovation Center for Cancer;
State Key Laboratory of Oncology in South China Collaborative Innovation Center for Cancer;
State Key Laboratory of Oncology in South China Collaborative Innovation Center for Cancer;
State Key Laboratory of Oncology in South China Collaborative Innovation Center for Cancer;
State Key Laboratory of Oncology in South China Collaborative Innovation Center for Cancer;
State Key Laboratory of Oncology in South China Collaborative Innovation Center for Cancer;
State Key Laboratory of Oncology in South China Collaborative Innovation Center for Cancer;
Department of RadiotherapyThe Affiliated Yantai Yuhuangding Hospital of Qingdao UniversityYantai;
State Key Laboratory of Oncology in South China Collaborative Innovation Center for Cancer;
triple‐negative breast cancer; CDCA7; EZH2; proliferation; metastasis;
机译:CDCA7的过度表达预测预后差和诱导ezH2介导的三阴性乳腺癌进展
机译:ANCCA / ATAD2过表达确定了预后不良的乳腺癌患者,可通过控制B-Myb和EZH2来驱动三阴性细胞的增殖和存活。
机译:miR-146a-5p的过度表达上调了三阴性乳腺癌细胞中的Lncrna Hotair,预测预后差
机译:从Lincrna-ROR / miR-145 / ARF6表达途径产生三维曲线和siRNA mRNA ARF6的二维曲目,参与三重阴性乳腺癌进展
机译:鉴定LRP8,CDCA7和MLK4作为三阴性乳腺癌中癌症干细胞的新疗法靶标
机译:EZH2 / NSD2组甲基转移酶轴的过度表达预测预后差加速了三阴性乳腺癌中的肿瘤进展
机译:EZH2 / NSD2组甲基转移酶轴的过度表达预测预后差,加速了三阴性乳腺癌中的肿瘤进展