TGF-beta 1 disturbs airway epithelia integrity and decreases claudin 3 in tight junctions via activating SMAD 2/3 proteins.

Schilpp C.; Lochbaum R.; Frick M.; Dietl P.; Wittekindt O.

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TGF-beta 1 disturbs airway epithelia integrity and decreases claudin 3 in tight junctions via activating SMAD 2/3 proteins.

机译：TGF-Beta 1通过激活Smad 2/3蛋白，扰动气道上皮细胞完整性并降低紧密交界中的克劳丁3。

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来源
《Acta physiologica》 |2019年第s719期|共1页
作者
Schilpp C.; Lochbaum R.; Frick M.; Dietl P.; Wittekindt O.;
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作者单位

Ulm Univ Inst Gen Physiol Ulm Germany;

Ulm Univ Inst Gen Physiol Ulm Germany;

Ulm Univ Inst Gen Physiol Ulm Germany;

Ulm Univ Inst Gen Physiol Ulm Germany;

Ulm Univ Inst Gen Physiol Ulm Germany;

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正文语种 eng
中图分类人体生理学;
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1. TGF-beta 1 disturbs airway epithelia integrity and decreases claudin 3 in tight junctions via activating SMAD 2/3 proteins. [J] . Schilpp C., Lochbaum R., Frick M., Acta physiologica . 2019,第S719期

机译：TGF-Beta 1通过激活Smad 2/3蛋白，扰动气道上皮细胞完整性并降低紧密交界中的克劳丁3。
2. MMP9 modulates tight junction integrity and cell viability in human airway epithelia [J] . Paola D. Vermeer, James Denker, Miriam Estin, American Journal of Physiology . 2009,第5aPta1期

机译：MMP9在人体气道上皮内调节紧密的结完整性和细胞活力
3. K deficiency caused defects in renal tubular cell proliferation, oxidative stress response, tissue repair and tight junction integrity, but enhanced energy production, proteasome function and cellular K+ uptakeproliferation, oxidative stress response, tissuerepair and tight junction integrity, but enhancedenergy production, proteasome function andcellular+ deficiency caused defects in renal tubular cellproliferation, oxidative stress response, tissuerepair and tight junction integrity, but enhancedenergy production, proteasome function andcellular K+ uptakeK deficiency caused defects in renal tubular cell proliferation, oxidative stress response, tissue repair and tight junction integrity, but enhanced energy production, proteasome function and cellular Ksup xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xmlns:oasis="http://docs.oasis-open.orgs/oasis-exchange/table"+/sup uptake [J] . Chompunoot Kapincharanon, Visith Thongboonkerd Cell adhesion & migration . 2018,第3期

机译：钾缺乏引起肾小管细胞增殖，氧化应激反应，组织修复和紧密连接完整性方面的缺陷，但增强了能量产生，蛋白酶体功能和细胞对K +的吸收，氧化应激反应，组织修复和紧密连接完整性，但增强了能量产生，蛋白酶体功能和细胞+缺乏会导致肾小管细胞增殖，氧化应激反应，组织修复和紧密连接完整性方面的缺陷，但能量产生，蛋白酶体功能和细胞对K +摄取的增加会导致肾小管细胞增殖，氧化应激反应，组织修复和紧密连接完整性方面的缺陷，但能量增加生产，蛋白酶体功能和细胞K ^{+ 吸收}
4. A key sequence in the claudin-1 protein regulates tight junction structure andfunction [C] . Randall Mrsny, G. Thomas Brown, Jeffrey Tom, Controlled Release Society annual meeting . 2004

机译：claudin-1蛋白中的关键序列调节紧密连接的结构和功能
5. The role of tight junction proteins claudin-3 and claudin-7 in ureteric bud branching [D] . Haddad, Nicholas 2009

机译：紧密连接蛋白claudin-3和claudin-7在输尿管芽分支中的作用
6. MMP9 modulates tight junction integrity and cell viability in human airway epithelia [O] . Paola D. Vermeer, James Denker, Miriam Estin, -1

机译：MMP9调节人气道上皮细胞的紧密连接完整性和细胞活力
7. MMP9 modulates tight junction integrity and cell viability in human airway epithelia [O] . Vermeer, Paola D., Denker, James, Estin, Miriam, 2009

机译：MMP9调节人气道上皮细胞的紧密连接完整性和细胞活力

TGF-beta 1 disturbs airway epithelia integrity and decreases claudin 3 in tight junctions via activating SMAD 2/3 proteins.

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