机译:Petzold等人对文章的回应,“Rivaroxaban通过蛋白酶活化受体-1的抑制FXA驱动的血小板活化来降低动脉血栓形成”
German Centre for Cardiovascular Research Partner Site Munich Heart Alliance Germany (T.P. M.T.;
German Centre for Cardiovascular Research Partner Site Munich Heart Alliance Germany (T.P. M.T.;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
Medizinische Klinik und Poliklinik I Klinikum der Universit?t München (T.P. M.T. Z.Z. R.R. J.P;
Medizinische Klinik und Poliklinik I Klinikum der Universit?t München (T.P. M.T. Z.Z. R.R. J.P;
German Centre for Cardiovascular Research Partner Site Munich Heart Alliance Germany (T.P. M.T.;
German Centre for Cardiovascular Research Partner Site Munich Heart Alliance Germany (T.P. M.T.;
German Centre for Cardiovascular Research Partner Site Munich Heart Alliance Germany (T.P. M.T.;
German Centre for Cardiovascular Research Partner Site Munich Heart Alliance Germany (T.P. M.T.;
German Centre for Cardiovascular Research Partner Site Munich Heart Alliance Germany (T.P. M.T.;
St. Mary’s Square Institute for Vascular Surgery and Phlebology;
German Heart Center Institute for Laboratory Medicine Technical University Munich (F.R.);
Medizinische Klinik und Poliklinik I Klinikum der Universit?t München (T.P. M.T. Z.Z. R.R. J.P;
Medizinische Klinik und Poliklinik I Klinikum der Universit?t München (T.P. M.T. Z.Z. R.R. J.P;
Department of Anesthesiology Cell Therapeutics and Hemostaseology (M.S.) Ludwig-Maximilians;
Helmholtz Zentrum München Institute of Lung Biology and Disease Group Systems Medicine of Chronic;
Helmholtz Zentrum München Institute of Lung Biology and Disease Group Systems Medicine of Chronic;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
Institute of Transplantation Diagnostics and Cell Therapeutics Heinrich Heine University Medical;
Institute of Pathophysiology West German Heart and Vascular Center University Hospital Essen;
Institute of Pharmacology and Clinical Pharmacology (T. Hohlfeld) Medical Faculty of the Heinrich;
Cardiovascular Research Institute Düsseldorf Germany (L.D. P.M. C.H. A. Ayhan R.M. A;
German Centre for Cardiovascular Research Partner Site Munich Heart Alliance Germany (T.P. M.T.;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
From the Department of Cardiology Pulmonology and Vascular Medicine (L.D. P.M. C.H. A. Ayhan R;
机译:Petzold等人对文章的回应,“Rivaroxaban通过蛋白酶活化受体-1的抑制FXA驱动的血小板活化来降低动脉血栓形成”
机译:通过Violi等人对物品来说,“Rivaroxaban通过蛋白酶活化受体-1的抑制FXA驱动的血小板活化来降低动脉血栓形成。
机译:Rivaroxaban通过蛋白酶活化受体-1抑制FXA驱动的血小板活化来降低动脉血栓形成
机译:凝集素样氧化低密度脂蛋白受体-1(LOX-1)在体外流量下对人脑微血管内皮细胞的腺苷二磷酸活性血小板粘附的作用
机译:因子XIa(FXIa)催化域的分子相互作用:底物IX和S-2366的催化,蛋白酶Nexin 2(PN2)的Kunitz蛋白酶抑制域(KPI)的抑制以及与活化血小板表面的结合。
机译:胰蛋白酶样血小板蛋白酶传播蛋白酶激活的受体1裂解和血小板激活。
机译:Petzold等人对文章的回应,“Rivaroxaban通过蛋白酶活化受体-1的抑制FXA驱动的血小板活化来降低动脉血栓形成”