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机译:Ca2 + -calmodulin和Kv7通道近端C末端的浸渍相互作用
Tel Aviv Univ Sackler Fac Med Dept Physiol &
Pharmacol IL-69978 Tel Aviv Israel;
Tel Aviv Univ Sackler Fac Med Dept Physiol &
Pharmacol IL-69978 Tel Aviv Israel;
Tel Aviv Univ Inst Struct Biol Dept Biochem &
Mol Biol George S Wise Fac Life Sci Tel Aviv Israel;
Northeastern Univ Dept Pharmaceut Sci Boston MA 02115 USA;
Tel Aviv Univ Dept Physiol &
Pharmacol Lab Struct Physiol Sackler Fac Med Tel Aviv Israel;
Tel Aviv Univ Sackler Fac Med Dept Physiol &
Pharmacol IL-69978 Tel Aviv Israel;
Tel Aviv Univ Dept Physiol &
Pharmacol Lab Struct Physiol Sackler Fac Med Tel Aviv Israel;
Tel Aviv Univ Dept Physiol &
Pharmacol Lab Struct Physiol Sackler Fac Med Tel Aviv Israel;
Northeastern Univ Dept Pharmaceut Sci Boston MA 02115 USA;
Tel Aviv Univ Inst Struct Biol Dept Biochem &
Mol Biol George S Wise Fac Life Sci Tel Aviv Israel;
Tel Aviv Univ Sackler Fac Med Dept Physiol &
Pharmacol IL-69978 Tel Aviv Israel;
calmodulin; Kv7; KCNQ; Potassium channel; PIP2;
机译:在Kv7通道近端C末端的Ca2 +-钙调蛋白和PIP2相互作用
机译:Kv7通道的C端:多功能模块。
机译:奎纳克林抑制Kir通道的机制:Kir2.x通道的急性孔阻滞和PIP2与Kir2.x和Kir6.2通道相互作用的干扰。
机译:血管紧张素II受体的近端区域的束缚(AT_(1A)C-末端到细胞膜
机译:大鼠肺动脉kV7通道的药理学
机译:Ca2 +-钙调蛋白和PIP2在Kv7通道近端C末端的相互作用
机译:Kv7通道羧基末端与PIP2和钙调蛋白的竞争性相互作用