Urolithin A gains in antiproliferative capacity by reducing the glycolytic potential via the p53/TIGAR axis in colon cancer cells

Norden Elisabeth; Heiss Elke H.

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Urolithin A gains in antiproliferative capacity by reducing the glycolytic potential via the p53/TIGAR axis in colon cancer cells

机译：通过在结肠癌细胞中通过P53 / Tigar轴降低糖蛋白潜力，尿道蛋白在抗增殖能力中获得抗增殖能力

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摘要

Urolithin A inhibits growth of colon cancer cells alone and synergistically in combination with oxaliplatin. Those activities are markedly supported by activation of the p53/TIGAR axis and subsequent reduction of the cellular glycolytic potential by urolithin A.

机译：尿道素A抑制单独的结肠癌细胞的生长，并与Oxaliplatin组合协同效应。这些活性通过激活P53 / Tigar轴并随后通过Urokithin A减少细胞糖醇潜力。

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《Carcinogenesis》 |2019年第1期|共9页
作者
Norden Elisabeth; Heiss Elke H.;
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作者单位

Univ Vienna Dept Pharmacognosy Vienna Austria;

Univ Vienna Dept Pharmacognosy Vienna Austria;

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原文格式 PDF
正文语种 eng
中图分类肿瘤学;
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专利

1. Urolithin A gains in antiproliferative capacity by reducing the glycolytic potential via the p53/TIGAR axis in colon cancer cells [J] . Norden Elisabeth, Heiss Elke H. Carcinogenesis . 2019,第1期

机译：通过在结肠癌细胞中通过P53 / Tigar轴降低糖蛋白潜力，尿道蛋白在抗增殖能力中获得抗增殖能力
2. Phase-II metabolism limits the antiproliferative activity of urolithins in human colon cancer cells. [J] . Gonzalez-Sarrias A., Gimenez-Bastida J. A., Nunez-Sanchez M. A., European journal of nutrition . 2014,第3期

机译：II期代谢限制了尿石素在人结肠癌细胞中的抗增殖活性。
3. Antiproliferative effect of urolithin A, the ellagic acid-derived colonic metabolite, on hepatocellular carcinoma HepG2.2.15 cells by targeting Lin28a/let-7a axis [J] . Qiu Zhenpeng, Zhou Junxuan, Zhang Cong, Brazilian Journal of Medical and Biological Research . 2018,第7期

机译：尿石素A（鞣花酸衍生的结肠代谢产物）通过靶向Lin28a / let-7a轴对肝细胞癌HepG2.2.15细胞的抗增殖作用
4. Screening for Antiproliferative Effect of Lactobacillus Strains Against Colon Cancer HT-29 Cells [C] . Shumei Wang, Lanwei Zhang, Wei Gu, International Conference on Environment Materials and Environment Management . 2012

机译：乳酸杆菌菌株对结肠癌HT-29细胞的抗增殖作用的筛选
5. Potato and grape polyphenols, respectively, suppress high-fat diet-elevated oxidative stress/innate inflammation markers in porcine model and induce apoptosis in HCT-116 p53 +/+ and p53 -/- human colon cancer cell lines in vitro. [D] . Radhakrishnan, Sridhar. 2014

机译：马铃薯和葡萄中的多酚分别抑制猪模型中高脂饮食升高的氧化应激/先天性炎症标记，并在体外诱导HCT-116 p53 + / +和p53-/-人结肠癌细胞系中的凋亡。
6. Urolithin A gains in antiproliferative capacity by reducing the glycolytic potential via the p53/TIGAR axis in colon cancer cells [O] . Elisabeth Norden, Elke H Heiss -1

机译：尿石素A通过降低p53 / TIGAR轴在结肠癌细胞中的糖酵解潜能来获得抗增殖能力
7. Differential antiproliferative mechanisms of novel derivative of benzimidazo[1,2-alpha]quinoline in colon cancer cells depending on their p53 status [O] . Sedic, M, Poznic, M, Gehrig, P, 2008

机译：苯并咪唑[1,2-α]喹啉新衍生物在结肠癌细胞中的不同增殖抑制机制取决于其p53状态

Urolithin A gains in antiproliferative capacity by reducing the glycolytic potential via the p53/TIGAR axis in colon cancer cells

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