...
机译:MA24.09 Keap1 / NRF2和PI3K途径之间的协同作用用改变的新陈代谢驱动非小细胞肺癌
Acrf Stem Cells and Cancer Division The Walter and Eliza Hall Institute of Medical Research;
Metabolomics Australia;
Acrf Stem Cells and Cancer Division The Walter and Eliza Hall Institute of Medical Research;
Molecular Genetics of Cancer Division The Walter and Eliza Hall Institute of Medical Research;
Metabolomics Australia;
Metabolomics Australia;
Department of Anatomical Pathology St Vincent’s Hospital Melbourne;
Molecular Genetics of Cancer Division The Walter and Eliza Hall Institute of Medical Research;
Molecular Medicine Division The Walter and Eliza Hall Institute of Medical Research Melbourne;
Metabolomics Australia;
Acrf Stem Cells and Cancer Division The Walter and Eliza Hall Institute of Medical Research;
机译:MA24.09 Keap1 / NRF2和PI3K途径之间的协同作用用改变的新陈代谢驱动非小细胞肺癌
机译:Keap1 / NRF2和PI3K途径之间的协同作用将非小细胞肺癌与改变的免疫微环境驱动
机译:Keap1 / NRF2和PI3K途径之间的协同作用用改变的免疫微环境驱动非小细胞肺癌
机译:在非小肺癌细胞中的谱分析EGFR激酶抑制剂抵抗途径
机译:遗传变异对非小细胞肺癌(NSCLC)类花生酸代谢酶的影响
机译:通过KEAP1 / NRF2途径激活keap1驱动肺癌进展的功能突变失落
机译:Keap1 / NRF2和PI3K途径之间的协同作用用改变的免疫微环境驱动非小细胞肺癌