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机译:胰岛素通过在分化的3T3-L1 adipocytes中逆转ABCA1,ABCG1和SR-BI的上调,将MCP-1抑制MCP-1抑制胆固醇流出到HDL3 / APOA1
Sun Yat Sen Univ Sun Yat Sen Mem Hosp Guangzhou Guangdong Peoples R China;
Sun Yat Sen Univ Sun Yat Sen Mem Hosp Guangzhou Guangdong Peoples R China;
Sun Yat Sen Univ Affiliated Hosp 6 Guangzhou Guangdong Peoples R China;
Sun Yat Sen Univ Sun Yat Sen Mem Hosp Guangzhou Guangdong Peoples R China;
Sun Yat Sen Univ Sun Yat Sen Mem Hosp Guangzhou Guangdong Peoples R China;
机译:胰岛素通过在分化的3T3-L1 adipocytes中逆转ABCA1,ABCG1和SR-BI的上调,将MCP-1抑制MCP-1抑制胆固醇流出到HDL3 / APOA1
机译:13-羟基亚油酸可增加RAW264.7巨噬细胞中胆固醇转运蛋白ABCA1,ABCG1和SR-BI的表达并刺激apoA-I依赖性胆固醇外流
机译:MCP-1通过在HepG2细胞中通过PI3K / Akt翻译后调控来抑制ABCA1,ABCG1和SR-BI来影响RCT
机译:通过抑制ABCA1和ABCG1,砷抑制胆固醇流出
机译:13-羟基亚油酸可增加RAW264.7巨噬细胞中胆固醇转运蛋白ABCA1ABCG1和SR-BI的表达并刺激apoA-I依赖性胆固醇外流
机译:13-羟基亚油酸可增加RAW264.7巨噬细胞中胆固醇转运蛋白ABCA1,ABCG1和SR-BI的表达并刺激apoA-I依赖性胆固醇外流
机译:3-Deazaadenosine和胰岛素诱导3T3-L1成纤维细胞向脂肪细胞分化