FK506 Accelerates CoCl2-Induced Cytotoxicity via Endoplasmic Reticulum Stress Pathway in HepG2 Cells

Chung Sang Young; Choi Soo Jin Na

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FK506 Accelerates CoCl2-Induced Cytotoxicity via Endoplasmic Reticulum Stress Pathway in HepG2 Cells

机译：FK506通过HepG2细胞的内质网应激途径加速COCl2诱导的细胞毒性

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来源
《Transplantation: Official Journal of the Transplantation Society 》 |2018年第9suppla期| 共1页
作者
Chung Sang Young; Choi Soo Jin Na;
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作者单位

Chonnam Natl Univ Hosp Dept Surg Gwangju South Korea;

Chonnam Natl Univ Hosp Dept Surg Gwangju South Korea;

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原文格式 PDF
正文语种 eng
中图分类器官移植术 ;
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1. FK506 Accelerates CoCl2-Induced Cytotoxicity via Endoplasmic Reticulum Stress Pathway in HepG2 Cells [J] . Chung Sang Young, Choi Soo Jin Na Transplantation: Official Journal of the Transplantation Society . 2018 ,第9Suppla期

机译：FK506通过HepG2细胞的内质网应激途径加速COCl2诱导的细胞毒性
2. Licochalcone A induces apoptosis through endoplasmic reticulum stress via a phospholipase Cγ1-, Ca~(2+)-, and reactive oxygen species-dependent pathway in HepG2 human hepatocellular carcinoma cells [J] . A-Young Choi, Ji Hyun Choi, Keun-Young Hwang, Apoptosis: An international journal on programmed cell death . 2014 ,第4期

机译：Licochalcone A通过磷脂酶Cγ1-，Ca〜（2 +）-和反应性氧物种依赖性途径通过内质网应激诱导肝癌细胞凋亡。
3. Dengue 2 infection of HepG2 liver cells results in endoplasmic reticulum stress and induction of multiple pathways of cell death [J] . Chutima Thepparit, Atefeh Khakpoor, Sarawut Khongwichit, BMC research notes . 2013 ,第1期

机译：登革热2感染HepG2肝细胞导致内质网应激并诱导多种细胞死亡途径
4. Endoplasmic Reticulum Stress Signaling Pathways Is Involved in Trichosanthin-Induced Apoptosis in Human Cervical Cancer Cell Line Hela [C] . Huang Yiling, Huang Liming, You Chengcheng, 2010 4th International Conference on Bioinformatics and Biomedical Engineering . 2010

机译：内质网应激信号通路涉及天花粉蛋白诱导的人宫颈癌细胞株Hela的凋亡。
5. The calcium signaling pathway, endoplasmic reticulum stress, and programmed cell death in the yeast Saccharomyces cerevisiae [D] . Dudgeon, Drew D. 2008

机译：酿酒酵母中的钙信号通路，内质网应激和程序性细胞死亡
6. Glutathione S-Transferase P1 Protects Against Amodiaquine Quinoneimines-Induced Cytotoxicity but Does Not Prevent Activation of Endoplasmic Reticulum Stress in HepG2 Cells [O] . Yongjie Zhang, Shalenie P. den Braver-Sewradj, Michiel W. den Braver, 2018

机译：谷胱甘肽S-转移酶P1保护免受阿莫地喹喹醌引起的细胞毒性，但不能阻止内质网应激在HepG2细胞中的激活。
7. Role of the Death Receptor and Endoplasmic Reticulum Stress Signaling Pathways in Polyphyllin I-Regulated Apoptosis of Human Hepatocellular Carcinoma HepG2 Cells [O] . Qihui Luo, Dandan Yang, Qi Qi, 2018

机译：死亡受体和内质网胁迫信号传导途径在人肝细胞癌Hepg2细胞中的聚蛋白I调节凋亡中的作用

FK506 Accelerates CoCl2-Induced Cytotoxicity via Endoplasmic Reticulum Stress Pathway in HepG2 Cells

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