Short-term bed rest-induced insulin resistance cannot be explained by increased mitochondrial H2O2 emission

Dirks Marlou L.; Miotto Paula M.; Goossens Gijs H.; Senden Joan M.; Petrick Heather L.; van Kranenburg Janneau; van Loon Luc J. C.; Holloway Graham P.

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Short-term bed rest-induced insulin resistance cannot be explained by increased mitochondrial H2O2 emission

机译：短期床休息诱导的胰岛素抵抗不能通过增加的线粒体H 2 O 2发射来解释

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《The Journal of Physiology》 |2020年第1期|共15页
作者
Dirks Marlou L.; Miotto Paula M.; Goossens Gijs H.; Senden Joan M.; Petrick Heather L.; van Kranenburg Janneau; van Loon Luc J. C.; Holloway Graham P.;
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作者单位

Maastricht Univ NUTRIM Sch Nutr &

Translat Res Metab Med Ctr Maastricht Netherlands;

Univ Guelph Human Hlth &

Nutr Sci Guelph ON Canada;

Maastricht Univ NUTRIM Sch Nutr &

Translat Res Metab Med Ctr Maastricht Netherlands;

Maastricht Univ NUTRIM Sch Nutr &

Translat Res Metab Med Ctr Maastricht Netherlands;

Univ Guelph Human Hlth &

Nutr Sci Guelph ON Canada;

Maastricht Univ NUTRIM Sch Nutr &

Translat Res Metab Med Ctr Maastricht Netherlands;

Maastricht Univ NUTRIM Sch Nutr &

Translat Res Metab Med Ctr Maastricht Netherlands;

Univ Guelph Human Hlth &

Nutr Sci Guelph ON Canada;

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原文格式 PDF
正文语种 eng
中图分类人体生理学;
关键词
bed rest; insulin resistance; mitochondria; muscle disuse; reactive oxygen species;

机译：卧床休息;胰岛素抵抗;线粒体;肌肉剥离;活性氧;

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1. Short-term bed rest-induced insulin resistance cannot be explained by increased mitochondrial H2O2 emission [J] . Dirks Marlou L., Miotto Paula M., Goossens Gijs H., The Journal of Physiology . 2020,第1期

机译：短期床休息诱导的胰岛素抵抗不能通过增加的线粒体H 2 O 2发射来解释
2. Exercise recovery increases skeletal muscle H2O2 emission and mitochondrial respiratory capacity following two-weeks of limb immobilization [J] . Pileggi Chantal A., Hedges Christopher P., DSouza Randall F., Free Radical Biology and Medicine: The Official Journal of the Oxygen Society . 2018,第期

机译：在两周的肢体固定后，运动恢复增加了骨骼肌H2O2排放和线粒体呼吸能力
3. Impaired mitochondrial energy supply coupled to increased H2O2 emission under energy/redox stress leads to myocardial dysfunction during Type I diabetes [J] . Tocchetti Carlo G., Stanley Brian A., Sivakumaran Vidhya, Clinical Science . 2015,第7a8期

机译：线粒体能量供应受损，再加上能量/氧化还原应激下过氧化氢的释放会导致I型糖尿病期间的心肌功能障碍
4. Effects of a diet enriched in trans fatty acids (trans MUFA) on muscle mitochondrial functions and development of insulin resistance in rodents [C] . A.-L. Tardy, P. Rousset, C. Giraudet, International Symposium on Energy and Protein Metabolism and Nutrition . 2007

机译：富含反式脂肪酸（Trans Mufa）富含饮食对啮齿动物肌肉线粒体函数和胰岛素抵抗的影响的影响
5. Intracellular mechanisms involved in the induction of highly active antiretroviral therapy (HAART) - induced skeletal muscle insulin resistance and potential therapeutic approaches to increase insulin sensitivity. [D] . Bogachus, Lindsey Davis. 2011

机译：涉及诱导高活性抗逆转录病毒疗法（HAART）的细胞内机制-诱导骨骼肌胰岛素抵抗和提高胰岛素敏感性的潜在治疗方法。
6. Mitochondrial H2O2 emission and cellular redox state link excess fat intake to insulin resistance in both rodents and humans [O] . Ethan J. Anderson, Mary E. Lustig, Kristen E. Boyle, 2009

机译：啮齿动物和人类的线粒体H2O2排放和细胞氧化还原状态将过量的脂肪摄入与胰岛素抵抗联系起来
7. Short‐term bed rest‐induced insulin resistance cannot be explained by increased mitochondrial H2O2emission [O] . Marlou L. Dirks, Paula M. Miotto, Gijs H. Goossens, 2019

机译：短期床休息诱导的胰岛素抵抗不能通过增加的线粒体H2O2肿瘤来解释

Short-term bed rest-induced insulin resistance cannot be explained by increased mitochondrial H2O2 emission

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