机译:莫罗尼酰胺通过抑制NLRP3炎性激活来缓解Coxsackievirus B3诱导的心肌损伤细胞凋亡
Sichuan Univ West China Hosp Dept Cardiol 37 Guoxue Alley Chengdu 610041 Sichuan Peoples R China;
Sichuan Univ West China Hosp Dept Cardiol 37 Guoxue Alley Chengdu 610041 Sichuan Peoples R China;
North Sichuan Med Coll Dept Ophthalmol Affiliated Hosp Nanchong Sichuan Peoples R China;
North Sichuan Med Coll Dept Cardiol Affiliated Hosp Nanchong Sichuan Peoples R China;
North Sichuan Med Coll Dept Cardiol Affiliated Hosp Nanchong Sichuan Peoples R China;
North Sichuan Med Coll Dept Cardiol Affiliated Hosp 2 Nanchong Sichuan Peoples R China;
North Sichuan Med Coll Dept Cardiol Affiliated Hosp Nanchong Sichuan Peoples R China;
机译:莫罗尼酰胺通过抑制NLRP3炎性激活来缓解Coxsackievirus B3诱导的心肌损伤细胞凋亡
机译:抑制Calpain缓解Coxsackievirus B3诱导的心肌炎,通过抑制规范NLRP3炎症组/胱天蛋白酶-1介导和非甘露糖胱天蛋白酶-11介导的γ唑唑孔途径
机译:间充质基质细胞抑制了Coxsackeivirus B3诱导的炎症性心肌病模型中的NLRP3炎症体活化
机译:使用同位素编码的ATP - 亲和探针和靶向质谱法在NLRP3炎症组活化期间洞察Kinome扰动
机译:补体蛋白C1q调节巨噬细胞NLRP3炎性体激活。
机译:钙蛋白酶的抑制作用通过抑制经典的NLRP3炎症小体/ caspase-1介导的和非经典的caspase-11介导的凋亡途径来缓解柯萨奇B3诱导的心肌炎。
机译:莫罗尼酰胺通过抑制NLRP3炎性激活来缓解Coxsackievirus B3诱导的心肌损伤细胞凋亡