Berberine induces ZIP14 expression and modulates zinc redistribution to protect intestinal mucosal barrier during polymicrobial sepsis

He Yan; Yuan Xiaoming; Zuo Hao; Li Xiangwei; Sun Ying; Feng Aiwen

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Berberine induces ZIP14 expression and modulates zinc redistribution to protect intestinal mucosal barrier during polymicrobial sepsis

机译：小檗碱诱导ZIP14的表达，并调节锌再分分配，以保护肠道粘膜屏障在多发性脓毒症期间保护肠粘膜屏障

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Aims: The present study investigated if berberine might induce Zrt-Irt-like protein 14 (ZIP14) and affect zinc redistribution to protect intestinal barrier in sepsis.

机译：目的：目前的研究如果小檗碱可能诱导ZRT-IRT样蛋白14（ZIP14）并影响锌再分精，以保护肠脓毒症尿道屏障。

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来源
《Life sciences》 |2019年第2019期|共8页
作者
He Yan; Yuan Xiaoming; Zuo Hao; Li Xiangwei; Sun Ying; Feng Aiwen;
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作者单位

Nanjing Med Univ Affiliated Huaian Hosp 1 Dept Oncol Radiotherapy Huaian City Jiangsu Peoples;

Nanjing Med Univ Affiliated Huaian Hosp 1 Dept Gastrointestinal Surg Huaian City Jiangsu;

Nanjing Med Univ Affiliated Huaian Hosp 1 Dept Gastrointestinal Surg Huaian City Jiangsu;

Nanjing Med Univ Affiliated Huaian Hosp 1 Dept Gastrointestinal Surg Huaian City Jiangsu;

Nanjing Med Univ Affiliated Huaian Hosp 1 Dept Gastrointestinal Surg Huaian City Jiangsu;

Nanjing Med Univ Affiliated Huaian Hosp 1 Dept Gastrointestinal Surg Huaian City Jiangsu;

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原文格式 PDF
正文语种 eng
中图分类医药、卫生;
关键词
Berberine; ZIP14; Zinc redistribution; Intestinal mucosal barrier; Sepsis; IGF-I; Tight junction;

机译：小檗碱;ZIP14;锌再分配;肠粘膜屏障;败血症;IGF-I;紧密交界处;

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专利

1. Berberine induces ZIP14 expression and modulates zinc redistribution to protect intestinal mucosal barrier during polymicrobial sepsis [J] . He Yan, Yuan Xiaoming, Zuo Hao, Life sciences . 2019,第期

机译：小檗碱诱导ZIP14的表达，并调节锌再分分配，以保护肠道粘膜屏障在多发性脓毒症期间保护肠粘膜屏障
2. Berberine Prevents Intestinal Mucosal Barrier Damage During Early Phase of Sepsis in Rat through the Toll-Like Receptors Signaling Pathway [J] . Li Guo-xun, Wang Xi-mo, Jiang Tao, The Korean Journal of Physiology & Pharmacology . 2015,第1期

机译：小碱通过类似收费受体的信号传导通路，预防败血症早期大鼠肠粘膜屏障损伤
3. Berberine prevents damage to the intestinal mucosal barrier during early phase of sepsis in rat through mechanisms independent of the NOD-like receptors signaling pathway [J] . LiG.-X., WangX.-M., JiangT., European Journal of Pharmacology: An International Journal . 2014,第Null期

机译：小ber碱通过独立于NOD样受体信号传导途径的机制防止败血症早期对大鼠肠粘膜屏障的损害
4. RESEARCH ABOUT AMBIENT TEMPERATURE CHANGE INDUC DAMAGE OF SMALL INTESTINAL MUCOSAL IMMUNIZATION BARRIER IN RAT [C] . Fanggeng Wu, Qingxian Zhu, Guozhu Hu, The 13th International congress for stereology . 2011

机译：大鼠小肠粘膜免疫屏障环境温度变化诱发损伤的研究
5. Dectin-1 expression is altered by fungal infection, polymicrobial sepsis, and glucan administration. [D] . Ozment-Skelton, Tammy R. 2006

机译：Dectin-1的表达可通过真菌感染，细菌性败血症和葡聚糖给药而改变。
6. Glucose-Insulin-Potassium Alleviates Intestinal Mucosal Barrier Injuries Involving Decreased Expression of Uncoupling Protein 2 and NLR Family-Pyrin Domain-Containing 3 Inflammasome in Polymicrobial Sepsis [O] . Jun-liang Zhang, Yi-ting Chen, Guang-dao Chen, 2006

机译：葡萄糖-胰岛素-钾缓解了肠道黏膜屏障损伤，涉及解耦蛋白2和含NLR家族-蛋白结构域的3炎性体在多发性脓毒症中的表达减少。
7. Zinc dyshomeostasis during polymicrobial sepsis in mice involves zinc transporter Zip14 and can be overcome by zinc supplementation [O] . Weßels, Inga, Cousins, Robert J. 2015

机译：小鼠多发性败血症中的锌动态异常涉及锌转运蛋白Zip14，可通过补充锌来克服

Berberine induces ZIP14 expression and modulates zinc redistribution to protect intestinal mucosal barrier during polymicrobial sepsis

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