Syk negatively regulates TLR4-mediated IFN beta and IL-10 production and promotes inflammatory responses in dendritic cells

Yin Hui; Zhou Huaxin; Kang Yi; Zhang Xiaoju; Duan Xiaoxian; Alnabhan Ridab; Liang Shuang; Scott David A.; Lamont Richard J.; Shang Jia; Wang Huizhi

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Syk negatively regulates TLR4-mediated IFN beta and IL-10 production and promotes inflammatory responses in dendritic cells

机译：Syk负调节TLR4介导的IFNβ和IL-10的产生，并促进树突状细胞的炎症反应

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摘要

Background: While Syk has been shown to associate with TLR4, the immune consequences of Syk-TLR interactions and related molecular mechanisms are unclear.

机译：背景：尽管Syk已显示与TLR4相关，但Syk-TLR相互作用和相关分子机制的免疫后果尚不清楚。

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来源
《Biochimica et Biophysica Acta. General Subjects》 |2016年第3期|共11页
作者
Yin Hui; Zhou Huaxin; Kang Yi; Zhang Xiaoju; Duan Xiaoxian; Alnabhan Ridab; Liang Shuang; Scott David A.; Lamont Richard J.; Shang Jia; Wang Huizhi;
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正文语种 eng
中图分类生物化学;
关键词
Syk; Toll-like receptor 4; Interferon-beta; IL-10; PI3K; GSK3 beta;

机译：病;Toll样受体4;干扰素-β;IL-10;PI3K;GSK3β;

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专利

1. Syk negatively regulates TLR4-mediated IFN beta and IL-10 production and promotes inflammatory responses in dendritic cells [J] . Yin Hui, Zhou Huaxin, Kang Yi, Biochimica et Biophysica Acta. General Subjects . 2016,第3期

机译：Syk负调节TLR4介导的IFNβ和IL-10的产生，并促进树突状细胞的炎症反应
2. CD1d-Restricted IFN-γ–Secreting NKT Cells Promote Immune Complex-Induced Acute Lung Injury by Regulating Macrophage-Inflammatory Protein-1α Production and Activation of Macrophages and Dendritic Cells [J] . Ji Hyung Kim, Doo Hyun Chung The journal of immunology . 2011,第3期

机译：CD1d限制的IFN-γ分泌型NKT细胞通过调节巨噬细胞-炎性蛋白1α的产生以及巨噬细胞和树突状细胞的活化来促进免疫复合物诱导的急性肺损伤。
3. Defective Dendritic Cell Function in HIV-Infected Patients Receiving Effective Highly Active Antiretroviral Therapy: Neutralization of IL-10 Production and Depletion of CD4+CD25+ T Cells Restore High Levels of HIV-Specific CD4+ T Cell Responses Induced by Dendritic Cells Generated in the Presence of IFN-α [J] . Cédric Carbonneil, Vladimira Donkova-Petrini, Albertine Aouba, The journal of immunology . 2004,第12期

机译：在接受有效的高效抗逆转录病毒治疗的HIV感染患者中树突状细胞功能缺陷：中和IL-10的产生和CD4 + CD25 + T细胞的耗竭，可恢复高水平的HIV特异性CD4 + T细胞反应，这种反应是由存在以下情况的树突细胞诱导的干扰素
4. ALGINIC ACID OLIGOSACCHARIDE UP-REGULATES IFN-γ PRODUCTION BY LYMPH NODE CELLS [C] . Tadashi Yoshida, Aki Hirano, Hanae Wada, Annual Meeting of the Japanese Association for Animal Cell Technology . 2003

机译：含有淋巴结细胞的藻酸寡糖升高IFN-γ产生
5. The development of a cell culture process for the production of recombinant human beta inteferon (β-IFN) from a transfected Chinese hamster ovary (CHO) cell line [D] . Rodriguez, Jose Fernando 2008

机译：从转染的中国仓鼠卵巢（CHO）细胞系生产重组人β干扰素（β-IFN）的细胞培养方法的开发
6. Syk Negatively Regulates TLR4-mediated IFNβ and IL-10 production and Promotes Inflammatory Responses in Dendritic Cells [O] . Hui Yin, Zhou Huaxin, Yi Kang, -1

机译：Syk负调节树突状细胞中TLR4介导的IFNβ和IL-10的产生并促进炎症反应。
7. Syk negatively regulates TLR4-mediated IFNβ and IL-10 production and promotes inflammatory responses in dendritic cells [O] . Hui Yin, Huaxin Zhou, Yi Kang, 2016

机译：Syk负调节TLR4介导的IFNβ和IL-10产生，并促进树突细胞中的炎症反应

Syk negatively regulates TLR4-mediated IFN beta and IL-10 production and promotes inflammatory responses in dendritic cells

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