Polypeptide from Chlamys farreri suppresses ultraviolet-B irradiation-induced apoptosis through restoring ER redox homeostasis, scavenging ROS generation, and suppressing the PERK-eIF2a-CHOP pathway in HaCaT cells

Zhong Feng; Xie Jing; Zhang Di; Han Yantao; Wang Chunbo

首页> 外文期刊>Journal of Photochemistry and Photobiology, B. Biology: Official Journal of the European Society for Photobiology >Polypeptide from Chlamys farreri suppresses ultraviolet-B irradiation-induced apoptosis through restoring ER redox homeostasis, scavenging ROS generation, and suppressing the PERK-eIF2a-CHOP pathway in HaCaT cells

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Polypeptide from Chlamys farreri suppresses ultraviolet-B irradiation-induced apoptosis through restoring ER redox homeostasis, scavenging ROS generation, and suppressing the PERK-eIF2a-CHOP pathway in HaCaT cells

机译：来自衣原体的多肽通过恢复ER氧化还原稳态，清除ROS生成并抑制HaCaT细胞中的PERK-eIF2a-CHOP途径来抑制紫外线B辐射诱导的凋亡

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Objective: The aim of this study was to investigate the effect of polypeptide from Chlamys farreri (PCF) on ultraviolet B (UVB) irradiation-induced apoptosis in human keratinocyte HaCaT cells.

机译：目的：本研究的目的是研究费氏衣原体（PCF）多肽对紫外线B（UVB）辐射诱导的人角质形成细胞HaCaT细胞凋亡的影响。

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《Journal of Photochemistry and Photobiology, B. Biology: Official Journal of the European Society for Photobiology》 |2015年第null期|共7页
作者
Zhong Feng; Xie Jing; Zhang Di; Han Yantao; Wang Chunbo;
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正文语种 eng
中图分类生物科学;化学;
关键词
HaCaT cells; Ultraviolet-B (UVB) irradiation; Endoplasmic reticulum (ER); Unfolded protein response (UPR); PERK-eIF2a-CHOP pathway;

机译：HaCaT细胞;紫外线B（UVB）照射;内质网（ER）;未折叠蛋白反应（UPR）;PERK-eIF2a-CHOP途径;

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1. Polypeptide from Chlamys farreri suppresses ultraviolet-B irradiation-induced apoptosis through restoring ER redox homeostasis, scavenging ROS generation, and suppressing the PERK-eIF2a-CHOP pathway in HaCaT cells [J] . Zhong Feng, Xie Jing, Zhang Di, Journal of Photochemistry and Photobiology, B. Biology: Official Journal of the European Society for Photobiology . 2015,第Null期

机译：来自衣原体的多肽通过恢复ER氧化还原稳态，清除ROS生成并抑制HaCaT细胞中的PERK-eIF2a-CHOP途径来抑制紫外线B辐射诱导的凋亡
2. Polypeptide from Chlamys farreri restores endoplasmic reticulum (ER) redox homeostasis, suppresses ER stress, and inhibits ER stress-induced apoptosis in ultraviolet B-irradiated HaCaT cells [J] . Jing Xie, Feng Zhong, Yantao Han, American Journal of Translational Research . 2015,第5期

机译：法氏衣原体的多肽可恢复内质网（ER）氧化还原稳态，抑制ER应激并抑制ER应激诱导的紫外线B照射的HaCaT细胞凋亡
3. Polypeptide from Chlamys farreri restores endoplasmic reticulum (ER) redox homeostasis, suppresses ER stress, and inhibits ER stress-induced apoptosis in ultraviolet B-irradiated HaCaT cells [J] . Jing Xie, Feng Zhong, Yantao Han, American Journal of Translational Research . 2015,第5期

机译：法氏衣原体的多肽可恢复内质网（ER）氧化还原稳态，抑制ER应激并抑制ER应激诱导的紫外线B照射的HaCaT细胞凋亡
4. DLX4 homeoprotein promotes PI3K/Akt anti-apoptosis pathway in ER negative breast cancer cells through upregulation of VEGFA. [D] . Wu, Weiwei. 2010

机译：DLX4同源蛋白通过上调VEGFA促进ER阴性乳腺癌细胞中的PI3K / Akt抗凋亡途径。
5. Polypeptide from Chlamys farreri restores endoplasmic reticulum (ER) redox homeostasis suppresses ER stress and inhibits ER stress-induced apoptosis in ultraviolet B-irradiated HaCaT cells [O] . Jing Xie, Feng Zhong, Yantao Han, 2015

机译：来自衣原体的多肽可恢复内质网（ER）氧化还原稳态抑制ER应激并抑制ER应激诱导的紫外线B照射的HaCaT细胞凋亡
6. Chemopreventive mechanism of polypeptides from Chlamy Farreri (PCF) against UVB-induced malignant transformation of HaCaT cells [O] . Yantao Han, Huihui Zhao, Qixiao Jiang, 2014

机译：来自Chllamy Farreri（PCF）的多肽的化学预防机制免受UVB诱导的HACAT细胞恶性转化

Polypeptide from Chlamys farreri suppresses ultraviolet-B irradiation-induced apoptosis through restoring ER redox homeostasis, scavenging ROS generation, and suppressing the PERK-eIF2a-CHOP pathway in HaCaT cells

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