The Zinc Ion Chelating Agent TPEN Attenuates Neuronal Death/apoptosis Caused by Hypoxia/ischemia Via Mediating the Pathophysiological Cascade Including Excitotoxicity, Oxidative Stress, and Inflammation

Wang Wei-Ming; Liu Zhao; Liu Ai-Jun; Wang Yu-Xiang; Wang Hong-Gang; An Di; Heng Bin; Xie Lai-Hua; Duan Jun-Li; Liu Yan-Qiang

首页> 外文期刊>CNS neuroscience & therapeutics >The Zinc Ion Chelating Agent TPEN Attenuates Neuronal Death/apoptosis Caused by Hypoxia/ischemia Via Mediating the Pathophysiological Cascade Including Excitotoxicity, Oxidative Stress, and Inflammation

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The Zinc Ion Chelating Agent TPEN Attenuates Neuronal Death/apoptosis Caused by Hypoxia/ischemia Via Mediating the Pathophysiological Cascade Including Excitotoxicity, Oxidative Stress, and Inflammation

机译：锌离子螯合剂TPEN通过介导包括兴奋性毒性，氧化应激和炎症在内的病理生理级联减轻由缺氧/缺血引起的神经元死亡/凋亡。

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AimsWe aim to determine the significant effect of TPEN, a Zn2+ chelator, in mediating the pathophysiological cascade in neuron death/apoptosis induced by hypoxia/ischemia.

机译：目的我们旨在确定TPEN（一种Zn2 +螯合剂）在介导由缺氧/缺血引起的神经元死亡/凋亡的病理生理学级联中的重要作用。

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来源
《CNS neuroscience & therapeutics》 |2015年第9期|共10页
作者
Wang Wei-Ming; Liu Zhao; Liu Ai-Jun; Wang Yu-Xiang; Wang Hong-Gang; An Di; Heng Bin; Xie Lai-Hua; Duan Jun-Li; Liu Yan-Qiang;
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正文语种 eng
中图分类神经病学与精神病学;
关键词
Excitotoxicity; Free Zn2+; Hypoxia; ischemia; Inflammation; Oxidative stress; TPEN;

机译：兴奋毒性;游离锌离子;缺氧;缺血;炎症;氧化应激;TPEN;

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1. The Zinc Ion Chelating Agent TPEN Attenuates Neuronal Death/apoptosis Caused by Hypoxia/ischemia Via Mediating the Pathophysiological Cascade Including Excitotoxicity, Oxidative Stress, and Inflammation [J] . Wang Wei-Ming, Liu Zhao, Liu Ai-Jun, CNS neuroscience & therapeutics . 2015,第9期

机译：锌离子螯合剂TPEN通过介导包括兴奋性毒性，氧化应激和炎症在内的病理生理级联减轻由缺氧/缺血引起的神经元死亡/凋亡。
2. Electroacupuncture Attenuates Cerebral Ischemia and Reperfusion Injury in Middle Cerebral Artery Occlusion of Rat via Modulation of Apoptosis, Inflammation, Oxidative Stress, and Excitotoxicity [J] . Shen Mei-hong, Zhang Chun-bing, Zhang Jia-hui, Evidence-based complementary and alternative medicine: eCAM . 2016,第3期

机译：通过调节细胞凋亡，炎症，氧化应激和兴奋毒性，电针衰减大鼠中脑动脉闭塞的脑缺血和再灌注损伤
3. Resveratrol protects neuronal cells from isoflurane-induced inflammation and oxidative stress-associated death by attenuating apoptosis via Akt/p38 MAPK signaling [J] . Hu Weilan, Yang Ei, Ye Jianxin, Experimental and therapeutic medicine . 2018,第2aPta1期

机译：白藜芦醇通过AKT / P38 MAPK信号传导来保护来自异氟醚诱导的异氟烷诱导的炎症和氧化应激相关的死亡的神经元细胞
4. Role of ATF4 in neuronal death mediated by DNA damage, endoplasmic reticulum stress and ischemia-hypoxia. [D] . Galehdar, Zohreh. 2013

机译：ATF4在DNA损伤，内质网应激和缺血缺氧介导的神经元死亡中的作用。
5. The Zinc Ion Chelating Agent TPEN Attenuates Neuronal Death/apoptosis Caused by Hypoxia/ischemia Via Mediating the Pathophysiological Cascade Including Excitotoxicity Oxidative Stress and Inflammation [O] . Wei‐Ming Wang, Zhao Liu, Ai‐Jun Liu, 2015

机译：锌离子螯合剂TPEN通过介导包括兴奋性毒性氧化应激和炎症在内的病理生理级联减轻由缺氧/缺血引起的神经元死亡/凋亡。
6. The Zinc Ion Chelating Agent TPEN Attenuates Neuronal Death/apoptosis Caused by Hypoxia/ischemia Via Mediating the Pathophysiological Cascade Including Excitotoxicity, Oxidative Stress, and Inflammation [O] . Wei-Ming Wang, Zhao Liu, Ai-Jun Liu, 2015

机译：锌离子螯合剂TPEN通过介导病理生理学级联，包括吞噬毒性，氧化应激和炎症，衰减由缺氧/缺血引起的神经元死亡/凋亡

The Zinc Ion Chelating Agent TPEN Attenuates Neuronal Death/apoptosis Caused by Hypoxia/ischemia Via Mediating the Pathophysiological Cascade Including Excitotoxicity, Oxidative Stress, and Inflammation

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