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Mechanism of coronary malperfusion due to type-a aortic dissection.

机译:A型主动脉夹层引起的冠状动脉灌注不足的机制。

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摘要

A 44-year-old man was admitted to a community hospital with acute onset of chest pain. Based on ECG findings and a mild troponin elevation, the patient was initially treated for acute coronary syndrome. On the 2nd day of hospitalization, troponin increased to 12 ng/ml and transthoracic echocardiography showed severe aortic regurgita-tion, so that computed tomography was performed and revealed Stanford type-A aortic dissection of the ascending aorta. The patient was immediately transferred to the hybrid operating room of our hospital. Preoperative coronary angiography showed non-stenotic coronary heart disease. Intraoperative trans-esophageal echocardiography revealed an intimal flap beginning just above the aortic valve. During diastole, the intimal flap prolapsed through the aortic valve, resulting in intermittent diastolic occlusion of the coronary ostia (Figure 1). Emergency operation resulted in successful replacement of the ascending aorta and reconstruction of the aortic valve.
机译:一名44岁的男子因急性胸痛入院一家社区医院。基于心电图检查结果和轻度肌钙蛋白升高,该患者最初接受了急性冠脉综合征的治疗。住院第二天,肌钙蛋白升高至12 ng / ml,经胸超声心动图显示严重的主动脉瓣反流,因此进行了计算机断层扫描并显示了升主动脉的Stanford A型主动脉夹层。病人立即被转移到我们医院的混合手术室。术前冠状动脉造影显示非狭窄性冠心病。术中经食道超声心动图检查发现内膜瓣开始于主动脉瓣上方。舒张期,内膜瓣通过主动脉瓣脱垂,导致冠状动脉口的舒张期间歇性闭塞(图1)。紧急操作成功地替换了升主动脉并重建了主动脉瓣。

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