Capsular contracture and genetic profile of ica genes among Staphylococcus epidermidis isolates from subclinical periprosthetic infections.

摘要

We read with great interest the article by Tamboto, Vickery, and Deva in the September of 2010 issue of the Journal on the role of subclinical infection and biofilm in capsular fibrosis.1 The methods used and the animal model developed are very interesting and are certainly an approximation of use in humans. We are interested in the fact that for the first time someone has shown a direct causal link between the presence of biofilm and capsular contracture in vivo in an animal model. However, our experience in humans shows quite different results.