机译:International Congress on Hormonal Steroids and Hormones and Cancer: Loss of PTEN expression followed by Akt phosphorylation is a poor prognostic factor for patients with endometrial cancer.
机译:International Congress on Hormonal Steroids and Hormones and Cancer: Adaptive hypersensitivity to estrogen: mechanism for superiority of aromatase inhibitors over selective estrogen receptor modulators for breast cancer treatment and prevention.
机译:International Congress on Hormonal Steroids and Hormones and Cancer: Endocrine/paracrine/autocrine survival factor activity of bone microenvironment participates in the development of androgen ablation and chemotherapy refractoriness of prostate canc
机译:Endocrine/paracrine/autocrine survival factor activity of bone microenvironment participates in the development of androgen ablation and chemotherapy refractoriness of prostate cancer metastasis in skeleton.
机译:International Congress on Hormonal Steroids and Hormones and Cancer: Up-regulation of LRP16 mRNA by 17beta-estradiol through activation of estrogen receptor alpha (ERalpha), but not ERbeta, and promotion of human breast cancer MCF-7 cell proliferatio
机译:Adaptive hypersensitivity to estrogen: mechanism for superiority of aromatase inhibitors over selective estrogen receptor modulators for breast cancer treatment and prevention.
机译:International Congress on Hormonal Steroids and Hormones and Cancer: The role of vitamin D and retinoids in controlling prostate cancer progression.
机译:International Congress on Hormonal Steroids and Hormones and Cancer: The expression and function of estrogen receptor alpha and beta in human breast cancer and its clinical application.
机译:International Congress on Hormonal Steroids and Hormones and Cancer: Potential role of gonadotrophin-releasing hormone (GnRH)-I and GnRH-II in the ovary and ovarian cancer.
机译:International Congress on Hormonal Steroids and Hormones and Cancer: The angiogenic factor CYR61 in breast cancer: molecular pathology and therapeutic perspectives.
机译:Up-regulation of LRP16 mRNA by 17beta-estradiol through activation of estrogen receptor alpha (ERalpha), but not ERbeta, and promotion of human breast cancer MCF-7 cell proliferation: a preliminary report.