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机译:硫柳汞诱导的人SCM1胃癌细胞凋亡:p38 MAP激酶和Caspase-3途径的激活而没有涉及[Ca 2 + sup>] i sub>升高
Department of Surgery Kaohsiung Veterans General Hospital Kaohsiung Taiwan 813;
Department of Nursery Tzu Hui Institute of Technology Pingtung Taiwan 926;
Department of Psychiatry Kaohsiung Medical University Hospital Kaohsiung Taiwan 807;
Department of Psychiatry Tian-Sheng Memorial Hospital Ping-Tong Taiwan 900;
Laboratory Medicine Division Zuoying Armed Forces General Hospital Kaohsiung Taiwan 813;
Department of Surgery Ping Tung Christian Hospital Ping Tung Taiwan 900;
Department of Rehabilitation Kaohsiung Veterans General Hospital Kaohsiung Taiwan 813;
Department of Orthopaedic Surgery Kaohsiung Veterans General Hospital Kaohsiung Taiwan 813;
Department of Otolaryngology Kaohsiung Veterans General Hospital Kaohsiung Taiwan 813;
Department of Medical Education and Research Kaohsiung Veterans General Hospital Kaohsiung Taiwan 813;
机译:硫柳汞诱导人SCM1胃癌细胞凋亡:激活p38 MAP激酶和caspase-3途径,而无(Ca2 +)i升高。
机译:帕罗西汀诱导的人骨肉瘤细胞凋亡:激活p38 MAP激酶和caspase-3途径,而无(Ca2 +)i升高。
机译:Desipramine诱导的人PC3前列腺癌细胞凋亡:JNK激酶和caspase-3途径的激活和(Ca2 +)i升高的保护作用。
机译:抗磷脂综合征中NFKB活化的调节:ERK1 / 2和P38映射激酶途径的累积
机译:通过癌细胞中的RAS-MAPK途径研究诱导型丝裂原和应激激活的蛋白激酶1(MSK1)和组蛋白H3磷酸化。
机译:二烯丙基二硫键诱导的人胃癌MGC803细胞G2 / M阻滞涉及p38 MAP激酶途径的激活
机译:人SCM1胃癌细胞中的嗜鼻凋亡:P38 MAP激酶和Caspase-3途径的激活而不参与Ca2 + i升降