机译:NGF-TrkA介导的交感神经元转录因子CREB的逆行信号
Department of Neuroscience, Johns Hopkins University School of Medicine, 725 North Wolfe Street, Baltimore, MD 21205, USA.;
Animals; Animals; Newborn; Axonal Transport; Axons metabolism; Carbazoles pharmacology; Cell Membrane metabolism; Cells; Cultured; DNA-Binding Protein; Cyclic AMP-Responsive metabolism; Microspheres; Nerve Growth Factors metabolism pharmacology; Neurons metabolism; Phosphorylation; Proto-Oncogene Proteins antagonists inhibitors metabolism; Rats; Receptor Protein-Tyrosine Kinases antagonists inhibitors metabolism; Receptor; trkA; Receptors; Nerve Growth Factor antagonists inhibitors metabolism; Research Support; Non-U.S. Gov't; Signal Transduction; Superior Cervical Ganglion cytology;
机译:交感神经元中转录因子CREB的特异性切除令人惊讶地保护其免受发育调节的细胞凋亡。
机译:由交感神经元依赖NGF生存的CREB家庭转录因子介导。
机译:Pea3转录因子家族成员Etv4和Etv5介导NGF响应的DRG感觉神经元的逆行信号和轴突生长
机译:CREB结合蛋白(CBP)p300增强心肌相关转录因子-A(MRTF-A)激活的心肌肥大基因的表达。
机译:神经生长因子的逆行信号传导机制调节交感神经元的存活和凋亡。
机译:Pea3转录因子家族成员Etv4和Etv5介导响应NGF的DRG感觉神经元的逆行信号和轴突生长。
机译:在交感神经元中的转录因子Creb的特异性消融令人惊讶地保护作用调节的细胞凋亡