机译:JNK信号转导途径的细胞质抑制剂
Howard Hughes Medical Institute and Program in Molecular Medicine, Department of Biochemistry and Molecular Biology, University of Massachusetts Medical School, 373 Plantation Street, Worcester, MA 01605, USA.;
Animals; COS Cells; Ca(2+)-Calmodulin Dependent Protein Kinase metabolism; Carrier Proteins chemistry metabolism; Cell Nucleus metabolism; Cell Transformation, Neoplastic; Cells, Cultured; Cloning, Molecular; Cytoplasm metabolism; DNA-Binding Protein, Cyclic AMP-Responsive metabolism; Fusion Proteins, bcr-abl metabolism; Gene Expression Regulation; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinase 9; Mitogen-Activated Protein Kinases; Molecular Sequence Data; Phosphorylation; Protein Kinases metabolism; Proto-Oncogene Proteins c-jun metabolism; Recombinant Fusion Proteins metabolism; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.; Signal Transduction; Trans-Activation (Genetics); Transcription Factors metabolism; Transfection;
机译:JNK的小肽抑制剂通过抑制JNK信号通路来预防MPTP诱导的黑色素多巴胺能损伤
机译:垂体腺苷酸环化酶激活多肽(PACAP)通过抑制JNK / SAPK和p38信号转导通路来防止海马神经元凋亡。
机译:JunD通过JNK信号转导途径介导生存信号
机译:AcSDKP通过阻断ERK1 / 2和JNK途径的激活来抑制PDGF诱导的心脏成纤维细胞的增殖和胶原蛋白表达
机译:与视网膜母细胞瘤蛋白的相互作用促进了JNK / p38信号转导途径对转录因子ATF2的调节。
机译:细胞铁耗竭刺激JNK和p38 MAPK信号传导通路ASK1-硫氧还蛋白的解离和ASK1的激活
机译:JNK信号转导途径的细胞质抑制剂
机译:磷脂酶C依赖性过程的aminosteroid抑制剂对TCR介导的人T细胞信号转导途径的影响