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机译:GDNF基因疗法可保护多巴胺能神经元免于变性
Department of Neurobiology and Anatomy, University of Rochester, Box 603, 601 Elmwood Avenue, Rochester, NY 14642, USA.;
Adenoviridae genetics; Animals; Corpus Striatum metabolism pathology; Dopamine physiology; Gene Expression; Gene Therapy; Genetic Vectors; Humans; Male; Molecular Sequence Data; Nerve Degeneration; Nerve Growth Factors; Nerve Tissue Proteins genetics; Neurons pathology physiology; Neuroprotective Agents; Oxidopamine; PC12 Cells; Parkinson Disease pathology therapy; Rats; Rats; Inbred F344; Research Support; Non-U.S. Gov't; Research Support; U.S. Gov't; P.H.S.; Substantia Nigra metabolism pathology; Transgenes;
机译:HSV介导的基因转移传递的Bcl-2和GDNF可以加成保护多巴胺能神经元免受6-OHDA诱导的变性。
机译:GDNF保护的中脑多巴胺能神经元免于在成人脑中由轴切术引起的变性。
机译:胶质细胞源性神经营养因子(GDNF)基因传递保护多巴胺能终端免于变性。
机译:NADPH氧化酶及微胶质激活6-0HDA诱导的多巴胺能神经元变性
机译:UCH11启动子标记下的EGFP表达皮质脊髓运动神经元和ALS小鼠模型中变性抗性脊柱运动神经元的亚群
机译:螺旋神经节神经元被保护免受变性GDNF基因治疗
机译:帕金与GDNF / RET信号传导共同预防多巴胺能神经元变性