机译:T辅助1免疫需要CD4(+)T细胞中补体驱动的NLRP3炎性体活性
Kings Coll London, Div Transplant Immunol & Mucosal Biol, MRC Ctr Transplantat, London SE1 9RT, England;
NHLBI, Lab Mol Immunol, Bldg 10, Bethesda, MD 20892 USA;
NHLBI, Lab Mol Immunol, Bldg 10, Bethesda, MD 20892 USA;
Univ Queensland, Inst Mol Biosci, Brisbane, Qld 4072, Australia;
Hannover Med Sch, Inst Med Microbiol & Hosp Epidemiol, D-30625 Hannover, Germany;
Hannover Med Sch, Inst Med Microbiol & Hosp Epidemiol, D-30625 Hannover, Germany;
Hannover Med Sch, Inst Med Microbiol & Hosp Epidemiol, D-30625 Hannover, Germany;
Univ Queensland, Inst Mol Biosci, Brisbane, Qld 4072, Australia;
NHLBI, Pathol Core, Bldg 10, Bethesda, MD 20892 USA;
Univ Dublin Trinity Coll, Sch Biochem & Immunol, Dublin 2, Ireland;
Univ Queensland, Inst Mol Biosci, Brisbane, Qld 4072, Australia;
NIAID, Parasit Dis Lab, Bethesda, MD 20892 USA;
NHLBI, Lab Mol Immunol, Bldg 10, Bethesda, MD 20892 USA;
Med Univ Lubeck, Inst Syst Inflammat Res, D-23538 Lubeck, Germany|Cincinnati Childrens Hosp Med Ctr, Div Immunobiol, Cincinnati, OH 45229 USA|Univ Cincinnati, Coll Med, Cincinnati, OH USA;
Univ Sheffield, Dept Infect & Immun, Sheffield S10 2RX, S Yorkshire, England;
Univ Queensland, Inst Mol Biosci, Brisbane, Qld 4072, Australia;
Kings Coll London, Fac Life Sci & Med, Genom Ctr, London SE1 9NH, England;
Kings Coll London, Div Transplant Immunol & Mucosal Biol, MRC Ctr Transplantat, London SE1 9RT, England|NIAMSD, Lymphocyte Cell Biol Sect, Mol Immunol & Inflammat Branch, Bethesda, MD 20892 USA;
UCL, Div Med, UK Natl Amyloidosis Ctr, Royal Free Campus, London NW3 2PF, England;
Kings Coll London, Div Immunol Infect & Inflammatory Dis, Acad Dept Rheumatol, London SE1 1UL, England;
NIAID, Lab Clin Infect Dis, Inflammat & Innate Immun Unit, 9000 Rockville Pike, Bethesda, MD 20892 USA;
Kings Coll London, Div Transplant Immunol & Mucosal Biol, MRC Ctr Transplantat, London SE1 9RT, England|NHLBI, Lab Mol Immunol, Bldg 10, Bethesda, MD 20892 USA;
机译:幽门螺杆菌诱导的先天免疫细胞中的IL-1β分泌受NLRP3炎性小体的调节,并需要cag致病岛
机译:幽门螺杆菌诱导的先天免疫细胞中的IL-1β分泌受NLRP3炎性小体的调节,并需要cag致病岛
机译:幽门螺杆菌诱导的先天免疫细胞IL-1β分泌受NLRP3炎性小体调控,需要Cag致病岛
机译:CD4 + T辅助细胞依赖癌基因灭活消退的肿瘤免疫数学模型
机译:CD4 + T细胞在抗病毒免疫中的功能异质性和辅助作用。
机译:T辅助1免疫需要CD4 + T细胞中补体驱动的NLRP3炎性体活性
机译:T Helper 1免疫力需要在CD4 + T细胞中补充驱动的NLRP3炎性活性