机译:CRM1通过Leptomycin B的共价结合和水解抑制核输出
Departments of Pharmacology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9041;
Departments of Biochemistry, University of Texas Southwestern Medical Center, Dallas, TX 75390-9041;
Departments of Biochemistry, University of Texas Southwestern Medical Center, Dallas, TX 75390-9041;
Departments of Biochemistry, University of Texas Southwestern Medical Center, Dallas, TX 75390-9041;
Departments of Biochemistry, University of Texas Southwestern Medical Center, Dallas, TX 75390-9041;
Departments of Biochemistry, University of Texas Southwestern Medical Center, Dallas, TX 75390-9041;
Departments of Pharmacology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9041;
exportin-1; Xpo-1; lactone hydrolysis; michael addition; KPT inhibitor;
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机译:CRM1 / Ran介导的p27(Kip1)核出口涉及核出口信号,并将p27出口与蛋白水解联系起来。
机译:基于MS的CRM1出口途径和RANBP2 / NUP358在核细胞膜运输中的数量分析
机译:CRM1(出口蛋白1)抑制剂Leptomycin B和姜黄素,改变CRM1的亚细胞分布。
机译:CRM1通过Leptomycin B的共价结合和水解抑制核输出
机译:人CRM1的百霉素B敏感同源物促进核出口核蛋白质胰腺炎细胞的核出口