机译:在不同疾病模型中,铁递他汀类药物抑制氧化脂质损伤和细胞死亡
Department of Biological Sciences, Columbia University, 550 West 120th Street Northwest Corner Building, MC 4846 New York, New York 10027, United States;
Department of Biological Sciences, Columbia University, 550 West 120th Street Northwest Corner Building, MC 4846 New York, New York 10027, United States;
Department of Neurology, Children's Hospital and Harvard Medical School, Boston, Massachusetts 02115, United States;
Center for Drug Discovery and Department of Neurobiology, Duke University Medical Center, Durham, North Carolina 27704, United States;
Department of Biological Sciences, Columbia University, 550 West 120th Street Northwest Corner Building, MC 4846 New York, New York 10027, United States;
Department of Biological Sciences, Columbia University, 550 West 120th Street Northwest Corner Building, MC 4846 New York, New York 10027, United States;
Department of Neurology, Children's Hospital and Harvard Medical School, Boston, Massachusetts 02115, United States;
Center for Drug Discovery and Department of Neurobiology, Duke University Medical Center, Durham, North Carolina 27704, United States;
Department of Internal Medicine/Nephrology, University of Michigan, Ann Arbor, Michigan 48109, United States;
Clinic for Nephrology and Hypertension, Christian-Albrechts-University, Kiel 24105, Germany;
Department of Biological Sciences, Columbia University, 550 West 120th Street Northwest Corner Building, MC 4846 New York, New York 10027, United States,Department of Chemistry, Columbia University, New York, New York 10027, United States,Department of Systems Biology Columbia University, New York, New York 10027, United States,Howard Hughes Medical Institute, Columbia University, New York, New York 10027, United States;
机译:关于Ferrostatin-1和Liproxstatin-1的细胞保护机制以及脂质过氧化作用在肥大细胞死亡中的作用
机译:柑橘菌结合对帕金森病抑制氧化损伤和自噬相关细胞死亡的保护作用
机译:糖鞘脂溶酶体贮积病的多种小鼠模型中不变的自然杀伤T细胞的选择受损
机译:转导的Tat-DJ-1蛋白可防止氧化应激诱导的SH-SY5Y细胞死亡和帕金森氏病小鼠模型
机译:镰状细胞病中的脂质过氧化作用:血浆中无细胞血红蛋白的贡献。
机译:铁制他汀类抑制氧化脂质损伤和细胞多种疾病模型中的死亡
机译:Ferrostatins在不同疾病模型中抑制氧化脂损伤和细胞死亡