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机译:瘦素激活人类B细胞通过JAK2 / STAT3和p38MAPK / ERK1 / 2信号通路分泌TNF-α,IL-6和IL-10
Laboratories of Cellular and Molecular Immunology, Division of Basic and Clinical Immunology, University of California, Irvine, CA, 92697, USA;
Laboratories of Cellular and Molecular Immunology, Division of Basic and Clinical Immunology, University of California, Irvine, CA, 92697, USA;
Laboratories of Cellular and Molecular Immunology, Division of Basic and Clinical Immunology, University of California, Irvine, CA, 92697, USA;
Laboratories of Cellular and Molecular Immunology, Division of Basic and Clinical Immunology, University of California, Irvine, CA, 92697, USA;
B cells; cytokines; activation markers;
机译:瘦素通过JAK2 / STAT3和p38MAPK / ERK1 / 2信号通路激活人类B细胞分泌TNF-α,IL-6和IL-10。
机译:通过激活RAGE和ERK1 / 2,P38和STAT3信号传导途径,先进的糖糖末端产品在骨细胞样MLO-Y4细胞中诱导骨细胞样MLO-Y4细胞的产生和凋亡
机译:前列腺素H2类似物U-46619通过激活p38MAPK和ERK1 / 2信号通路提高人诱导的多能干细胞向内皮细胞的分化效率
机译:α-S1-酪蛋白(CSN1S1)通过JAK2 / Stat5a信号传导途径在山羊乳状上皮细胞中抑制β-酪蛋白表达
机译:IL-6通过pStat3发出信号,以防止人类调节性T细胞产生功能性免疫抑制。
机译:瘦素激活人类B细胞通过JAK2 / STAT3和p38MAPK / ERK1 / 2信号通路分泌TNF-αIL-6和IL-10
机译:瘦素激活人类B细胞通过JAK2 / STAT3和p38MAPK / ERK1 / 2信号通路分泌TNF-α,IL-6和IL-10