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机译:Lats和CK1的协同磷酸化通过SCFβ-TRCP调节YAP稳定性
Department of Pharmacology and Moores Cancer Center, University of California at San Diego, La Jolla, California 92093-0815, USA;
Department of Pharmacology and Moores Cancer Center, University of California at San Diego, La Jolla, California 92093-0815, USA|Department of Biological Chemistry, University of Michigan, Ann Arbor, Michigan 48109, USA;
Department of Pharmacology and Moores Cancer Center, University of California at San Diego, La Jolla, California 92093-0815, USA;
Laboratory of Molecular Signaling, Division of Oral Biology and Medicine, University of California at Los Angeles School of Dentistry, Los Angeles, California 90095, USA;
Department of Pharmacology and Moores Cancer Center, University of California at San Diego, La Jolla, California 92093-0815, USA;
YAP; Hippo pathway; CK1; SCF; Lats; phosphodegron;
机译:Lats和CK1的协同磷酸化通过SCF(beta-TRCP)调节YAP稳定性。
机译:DNA损伤通过SCFβ-TrCPE3连接酶调节UHRF1稳定性
机译:DNA损伤通过SCFβ-TrCPE3连接酶调节UHRF1稳定性
机译:多面Lyapunov在浓度和反应坐标下对生化系统的结构稳定性起作用
机译:SCF-β-TRCPE3泛素连接酶复合物的底物:识别和传递到蛋白酶体的机制。
机译:Lats和CK1的协同磷酸化通过SCFβ-TRCP调节YAP稳定性
机译:Lats和CK1的协同磷酸化通过SCFβ-TRCP调节YAP稳定性
机译:鉴定小的非肽配体结合scf-β-TRCp泛素连接酶靶向ER以进行泛素化和降解