机译:过度的内质网应激和导致的自噬通量功能障碍导致氟化物诱导的神经毒性
Department of Environmental Health, MOE Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology;
Department of Environmental Health, MOE Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology;
Department of Environmental Health, MOE Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology;
Department of Environmental Health, MOE Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology;
Department of Environmental Health, MOE Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology;
Department of Environmental Health, MOE Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology;
Department of Environmental Health, MOE Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology;
Department of Environmental Health, MOE Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology;
Department of Environmental Health, MOE Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology;
Department of Environmental Health, MOE Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology;
NaF; Neurotoxicity; ER stress; Autophagy; Rats; SH-SY5Y cells;
机译:海藻糖通过选择性自噬刺激和自噬通量恢复改善骨关节炎发展中的氧化应激介导的线粒体功能障碍和内质网应激。
机译:海藻糖通过选择性自噬刺激和自噬通量恢复改善骨关节炎发展中的氧化应激介导的线粒体功能障碍和内质网应激。
机译:通过在糖尿病肾病中恢复自噬通量,抑制可溶性环氧化物水解酶衰减肾小管线粒体功能障碍和ER应力
机译:棕榈酸脂磷脂组合物的改变促进肝脂毒性的ER应激和细胞功能障碍
机译:黄嘌呤氧化酶有助于机械通气引起的diaphragm肌氧化应激和收缩功能障碍。
机译:海藻糖通过选择性自噬刺激和自噬通量恢复改善骨关节炎发展中的氧化应激介导的线粒体功能障碍和内质网应激。
机译:脂肪胰岛素抵抗力的自噬功能障碍与ER应激之间的互核相互作用