机译:非功能性垂体腺瘤中的PI3K / Akt / mTOR和Raf / MEK / ERK信号通路扰动
Institute of Endocrinology and Metabolism and Felsenstein Medical Research Center, Rabin Medical Center, Beilinson Campus and Sackler School of Medicine, Tel-Aviv University, Tel-Aviv, Israel;
Institute of Endocrinology and Metabolism and Felsenstein Medical Research Center, Rabin Medical Center, Beilinson Campus and Sackler School of Medicine, Tel-Aviv University, Tel-Aviv, Israel;
Non-functioning pituitary adenomas; Somatostatin; PI3K; mTOR; ERK;
机译:非功能性垂体腺瘤中的PI3K / Akt / mTOR和Raf / MEK / ERK信号通路扰动。
机译:一种新的硫化氢释放供体,HA-ADT,通过抑制PI3K / AKT / MTOR和RAS / RAF / MEK / ERK信号通路来抑制人乳腺癌细胞的生长
机译:信号中间体(PI3K / PTEN / AKT / mTOR和RAF / MEK / ERK途径)作为抗癌和抗血管生成治疗的治疗靶标
机译:在非功能性垂体腺瘤的分级立体定向放射后的长期随访中,体积MRI评估的肿瘤收缩
机译:FGF8通过MEK / ERK和PI3K / AKT信号转导级联调节新皮质原基中的模式和基因表达。
机译:人参皂苷Rh2通过调节小鼠TLR4 / PI3K / Akt / mTORRaf-1 / MEK / ERK和Keap1 / Nrf2 / HO-1信号通路来减轻脂多糖诱导的急性肺损伤
机译:垂体腺瘤中RAF / MEK / ERK和PI3K / AKT / mTOR通路的激活及其对下游效应子的影响。