机译:BAPTA和二甲基BAPTA通过钙螯合独立机制在3T3-L1脂肪细胞中的微管破坏。
Department of Cell Biology, Institute for Molecular and Cellular Regulation, Gunma University, 3-39-15 Showa-machi, Maebashi 371-8512, Japan;
Department of Cell Biology, Institute for Molecular and Cellular Regulation, Gunma University, 3-39-15 Showa-machi, Maebashi 371-8512, Japan;
Department of Cell Biology, Institute for Molecular and Cellular Regulation, Gunma University, 3-39-15 Showa-machi, Maebashi 371-8512, Japan;
Department of Cell Biology, Institute for Molecular and Cellular Regulation, Gunma University, 3-39-15 Showa-machi, Maebashi 371-8512, Japan;
Department of Cell Biology, Institute for Molecular and Cellular Regulation, Gunma University, 3-39-15 Showa-machi, Maebashi 371-8512, Japan;
Department of Cell Biology, Institute for Molecular and Cellular Regulation, Gunma University, 3-39-15 Showa-machi, Maebashi 371-8512, Japan;
insulin; calcium; glucose transporter; BAPTA; microtubule;
机译:BAPTA和二甲基BAPTA通过钙螯合独立机制在3T3-L1脂肪细胞中的微管破坏。
机译:BAPTA和二甲基BAPTA通过钙螯合独立机制在3T3-L1脂肪细胞中的微管破坏。
机译:BAPTA和EGTA对棕色脂肪细胞单通道Cl-电流的Ca2 +依赖性影响
机译:开发小卫星的Bapta机制
机译:5,5'-二硝基BAPTA和Mag-quin-1与镉,铅,锌,铜,镍和钙的二价阳离子的络合研究
机译:花粉管的生长与细胞外钙离子通量和细胞内钙梯度有关:BAPTA型缓冲液和高渗介质的作用。
机译:通过在3T3-L1 adipocytes中通过钙螯合螯合螯合螯合螯合物和二甲基Bapta与Bapta和二甲基Bapta中断