机译:骨骼肌特定的MKP-1删除揭示了调节肥胖诱导的胰岛素抵抗的p38 MAPK / JNK / Akt信号转导节点。
Department of Pharmacology, Yale University School of Medicine, New Haven, CT;
Department of Pharmacology, Yale University School of Medicine, New Haven, CT;
Department of Pharmacology, Yale University School of Medicine, New Haven, CT;
Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT,Program in Integrative Cell Signaling and Neurobiology of Metabolism, Yale University School of Medicine, New Haven, CT;
Cellular & Molecular Physiology and Department of Internal Medicine, Section of Endocrinology and Metabolism, Yale University School of Medicine, New Haven, CT;
Cellular & Molecular Physiology and Department of Internal Medicine, Section of Endocrinology and Metabolism, Yale University School of Medicine, New Haven, CT;
Department of Health and Kinesiology, Purdue University, West Lafayette, IN;
Department of Health and Kinesiology, Purdue University, West Lafayette, IN;
Cellular & Molecular Physiology and Department of Internal Medicine, Section of Endocrinology and Metabolism, Yale University School of Medicine, New Haven, CT;
Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT,Program in Integrative Cell Signaling and Neurobiology of Metabolism, Yale University School of Medicine, New Haven, CT;
Department of Pharmacology, Yale University School of Medicine, New Haven, CT,Department of Comparative Medicine, Yale University School of Medicine, New Haven, CT,Program in Integrative Cell Signaling and Neurobiology of Metabolism, Yale University School of Medicine, New Haven, CT;
机译:MKP-1的骨骼肌特异性缺失显示了调节肥胖诱导的胰岛素抵抗的P38 MAPK / JNK / AKT信号节点
机译:骨骼肌脂蛋白脂肪酶的特异性缺失可增强骨骼肌中的胰岛素信号传导,但会引起肝脏和其他组织的胰岛素抵抗
机译:Coriolus Versicolor含水提取物改善胰岛素抵抗与PI3K / AKT和P38 MAPK信号传导途径,参与糖尿病骨骼肌
机译:在人肺和皮肤成纤维细胞中研究了炎症和氧化应激信号转导中的MAPK ERK,JNK和P38。
机译:骨骼肌特异性MKP-1的缺失揭示了调节肥胖诱导的胰岛素抵抗的p38 MAPK / JNK / Akt信号传导节点
机译:MKP-1的骨骼肌特异性缺失显示了调节肥胖诱导的胰岛素抵抗的P38 MAPK / JNK / AKT信号节点