Morphine Postconditioning Protects Against Reperfusion Injury: the Role of Protein Kinase C-Epsilon, Extracellular Signal-Regulated Kinase 1/2 and Mitochondrial Permeability Transition Pores

Zuolei Chen; Donat R. Spahn; Xuewei Zhang; Yingzhi Liu; Haichen Chu; Zhongkai Liu

首页> 外文期刊>Cellular Physiology and Biochemistry >Morphine Postconditioning Protects Against Reperfusion Injury: the Role of Protein Kinase C-Epsilon, Extracellular Signal-Regulated Kinase 1/2 and Mitochondrial Permeability Transition Pores

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Morphine Postconditioning Protects Against Reperfusion Injury: the Role of Protein Kinase C-Epsilon, Extracellular Signal-Regulated Kinase 1/2 and Mitochondrial Permeability Transition Pores

机译：吗啡后处理可防止再灌注损伤：蛋白激酶C-Epsilon，细胞外信号调节激酶1/2和线粒体通透性过渡孔的作用

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《Cellular Physiology and Biochemistry》 |2016年第5期|共11页
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Zuolei Chen; Donat R. Spahn; Xuewei Zhang; Yingzhi Liu; Haichen Chu; Zhongkai Liu;
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中图分类细胞生物物理学;
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1. Sevoflurane postconditioning protects isolated rat hearts against ischemia-reperfusion injury: the role of radical oxygen species, extracellular signal-related kinases 1/2 and mitochondrial permeability transition pore [J] . Yun-Tai Yao, Li-Huan Li, Lei Chen, Molecular Biology Reports . 2010,第5期

机译：七氟醚后处理可保护离体大鼠心脏免受缺血-再灌注损伤：自由基氧，细胞外信号相关激酶1/2和线粒体通透性转换孔的作用
2. Sevoflurane postconditioning protects isolated rat hearts against ischemia-reperfusion injury: the role of radical oxygen species, extracellular signal-related kinases 1/2 and mitochondrial permeability transition pore [J] . Yao Yun-Tai, Li Li-Huan, Chen Lei, Molecular biology reports . 2010,第5期

机译：七氟醚后处理可保护离体大鼠心脏免于缺血再灌注损伤：自由基氧，细胞外信号相关激酶1/2和线粒体通透性转变孔的作用
3. Sevoflurane Postconditioning Protects Chronically-Infarcted Rat Hearts against Ischemia-Reperfusion Injury by Activation of Pro-survival Kinases and Inhibition of Mitochondrial Permeability Transition Pore Opening upon Reperfusion [J] . Yao YT, Li LH, Li LB, Biological & pharmaceutical bulletin . 2009,第11期

机译：七氟醚后处理可通过激活生存前激酶并抑制线粒体通透性转变孔在再灌注时开放来保护慢性梗塞大鼠心脏免受缺血再灌注损伤。
4. Activation of Androgen Receptor in Prostate Cancer: Role of Protein Kinase A and Extracellular Signal-regulated Kinases [C] . Yehia Daaka, Elizabeth Kasbohm, Charles Yowel International Symposium on Hormonal Carcinogenesis . 2005

机译：前列腺癌中雄激素受体的激活 - 蛋白激酶A和细胞外信号调节激酶的作用
5. RAF-1 kinase inhibitor protein-mediated cholecystokinin-2 receptor desensitization and extracellular signal-regulated kinase activation. [D] . Park, Jeseong. 2009

机译：RAF-1激酶抑制剂蛋白介导的胆囊收缩素2受体脱敏和细胞外信号调节激酶激活。
6. Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3β [O] . Guillaume Chanoit, SungRyul Lee, Jinkun Xi, -1

机译：外源锌通过使糖原合酶激酶3β失活靶向线粒体通透性过渡孔从而保护心肌细胞免受再灌注损伤
7. Morphine postconditioning protects against reperfusion injury: the role of protein kinase c-epsilon, extracellular signal-regulated kinase 1/2 and mitochondrial permeability transition pores [O] . Chen, Zuolei, Spahn, Donat R, Zhang, Xuewei, 2016

机译：吗啡后处理可防止再灌注损伤：蛋白激酶c-ε，细胞外信号调节激酶1/2和线粒体通透性转换孔的作用

Morphine Postconditioning Protects Against Reperfusion Injury: the Role of Protein Kinase C-Epsilon, Extracellular Signal-Regulated Kinase 1/2 and Mitochondrial Permeability Transition Pores

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