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机译:正常高氧血症改善脑血流量和氧合,并抑制实验性局灶性局部缺血的梗死周围去极化
Stroke and Neurovascular Regulation Laboratory Department of Radiology Massachusetts General Hospital Harvard Medical School Charlestown MA 02129;
Biomedical Engineering Department University of Texas at Austin Austin TX 78712;
Martinos Center for Biomedical Imaging Department of Radiology Massachusetts General Hospital Harvard Medical School Charlestown MA 02129;
Neuroprotection Research Laboratory Department of Radiology Massachusetts General Hospital Harvard Medical School Charlestown MA 02129 and;
Stroke Service and Neuroscience Intensive Care Unit Department of Neurology Massachusetts General Hospital Harvard Medical School Boston MA 02114 USA;
机译:正常高氧血症可改善脑血流量和氧合,并抑制实验性局灶性局部缺血的梗死周围去极化。
机译:正常高氧血症可改善脑血流量和氧合,并抑制实验性局灶性局部缺血的梗死周围去极化。
机译:在实验性局灶性脑缺血中,延迟高压氧合比早期长时间的正常血压高氧症更有效。
机译:大鼠大脑中动脉阻塞模型的梗塞核心和梗塞周围去极化的近红外漫反射成像
机译:短期常压高氧对严重颅脑损伤中局部脑组织氧合和脑脊液氧化应激指标的影响。
机译:正常高氧血症可改善脑血流量和氧合并抑制实验性局灶性局部缺血的梗死周围去极化
机译:normobaric hyperoxia改善脑血流量和氧合,并抑制实验局灶性缺血中的PERI-IMARCT去偏振
机译:正常高氧下清醒家兔的血气运输